p53-induced miR-15a/16-1 and AP4 form a double-negative feedback loop to regulate epithelial-mesenchymal transition and metastasis in colorectal cancer.
about
MicroRNA in pancreatic ductal adenocarcinoma and its precursor lesionsA microRNA code for prostate cancer metastasis.Emerging Non-Canonical Functions and Regulation by p53: p53 and StemnessRole of microRNA in epithelial to mesenchymal transition and metastasis and clinical perspectives.Aberrant Expression of MicroRNA-15a and MicroRNA-16 Synergistically Associates with Tumor Progression and Prognosis in Patients with Colorectal CancerFibroblasts from patients with major depressive disorder show distinct transcriptional response to metabolic stressors.Oncogenic Role of miR-15a-3p in 13q Amplicon-Driven Colorectal Adenoma-to-Carcinoma ProgressionIntegrating transcriptional and protein interaction networks to prioritize condition-specific master regulators.MicroRNA-21 regulates biological behavior by inducing EMT in human cholangiocarcinomaThe synergistic interaction between the calcineurin B subunit and IFN-γ enhances macrophage antitumor activity.MiR-16 regulates mouse peritoneal macrophage polarization and affects T-cell activation.Aqueous Oldenlandia diffusa extracts inhibits colorectal cancer cells via activating AMP-activated protein kinase signalings.Comparative genetic screens in human cells reveal new regulatory mechanisms in WNT signaling.Tumor suppressor p53 induces miR-15a processing to inhibit neuronal apoptosis inhibitory protein (NAIP) in the apoptotic response DNA damage in breast cancer cellMicroRNAs in the etiology of colorectal cancer: pathways and clinical implications.ZNF281/ZBP-99: a new player in epithelial-mesenchymal transition, stemness, and cancer.Oncogenic roles of EMT-inducing transcription factors.The p53/microRNA connection in gastrointestinal cancer.miRNA-15a/16: as tumor suppressors and more.p53 directly activates cystatin D/CST5 to mediate mesenchymal-epithelial transition: a possible link to tumor suppression by vitamin D3AP4 activates cell migration and EMT mediated by p53 in MDA-MB-231 breast carcinoma cells.MicroRNA-15a induces cell apoptosis and inhibits metastasis by targeting BCL2L2 in non-small cell lung cancer.MicroRNA-575 targets BLID to promote growth and invasion of non-small cell lung cancer cells.Involvement of Non-coding RNAs in the Signaling Pathways of Colorectal Cancer.Interplay Between Transcription Factors and MicroRNAs Regulating Epithelial-Mesenchymal Transitions in Colorectal Cancer.AP4 is required for mitogen- and c-MYC-induced cell cycle progression.Regulatory miRNAs in Colorectal Carcinogenesis and Metastasis.Regulation of activating protein-4-associated metastases of non-small cell lung cancer cells by miR-144.MiR-23b and miR-199a impair epithelial-to-mesenchymal transition during atrioventricular endocardial cushion formation.MicroRNAs as Regulator of Signaling Networks in Metastatic Colon Cancer.Triptolide exerts protective effects against fibrosis following ileocolonic anastomosis by mechanisms involving the miR-16-1/HSP70 pathway in IL-10-deficient mice.miR-1199-5p and Zeb1 function in a double-negative feedback loop potentially coordinating EMT and tumour metastasis.Up-regulation of CIT promotes the growth of colon cancer cells.The PRMT5/WDR77 complex regulates alternative splicing through ZNF326 in breast cancer.PDGFR-modulated miR-23b cluster and miR-125a-5p suppress lung tumorigenesis by targeting multiple components of KRAS and NF-kB pathways.A double-negative feedback loop between EpCAM and ERK contributes to the regulation of epithelial-mesenchymal transition in cancer.AP4 modulated by the PI3K/AKT pathway promotes prostate cancer proliferation and metastasis of prostate cancer via upregulating L-plastin.Differential cellular responses by oncogenic levels of c-Myc expression in long-term confluent retinal pigment epithelial cells.p53 and Mdm2 act synergistically to maintain cardiac homeostasis and mediate cardiomyocyte cell cycle arrest through a network of microRNAs.Modified miR-15a has therapeutic potential for improving treatment of advanced stage colorectal cancer through inhibition of BCL2, BMI1, YAP1 and DCLK1.
P2860
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P2860
p53-induced miR-15a/16-1 and AP4 form a double-negative feedback loop to regulate epithelial-mesenchymal transition and metastasis in colorectal cancer.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
p53-induced miR-15a/16-1 and A ...... tastasis in colorectal cancer.
@en
p53-induced miR-15a/16-1 and A ...... tastasis in colorectal cancer.
@nl
type
label
p53-induced miR-15a/16-1 and A ...... tastasis in colorectal cancer.
@en
p53-induced miR-15a/16-1 and A ...... tastasis in colorectal cancer.
@nl
prefLabel
p53-induced miR-15a/16-1 and A ...... tastasis in colorectal cancer.
@en
p53-induced miR-15a/16-1 and A ...... tastasis in colorectal cancer.
@nl
P2093
P1433
P1476
p53-induced miR-15a/16-1 and A ...... tastasis in colorectal cancer.
@en
P2093
Heiko Hermeking
Helge Siemens
Rene Jackstadt
Thomas Kirchner
P304
P356
10.1158/0008-5472.CAN-13-2203
P407
P577
2013-11-27T00:00:00Z