Stable expression of MutLγ in human cells reveals no specific response to mismatched DNA, but distinct recruitment to damage sites.
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New insights into the mechanism of DNA mismatch repairMlh2 is an accessory factor for DNA mismatch repair in Saccharomyces cerevisiae.Repeat instability during DNA repair: Insights from model systemsThe Repeat Expansion Diseases: The dark side of DNA repair.Mutation Processes in 293-Based Clones Overexpressing the DNA Cytosine Deaminase APOBEC3B.Association between DNA mismatch repair gene polymorphisms and platinum-based chemotherapy toxicity in non-small cell lung cancer patientsDigital gene expression profiling analysis of DNA repair pathways in colon cancer stem population of HT29 cells.A spontaneous mutation in MutL-Homolog 3 (HvMLH3) affects synapsis and crossover resolution in the barley desynaptic mutant des10.Localization of MLH3 at the centrosomesThe MLH1 ATPase domain is needed for suppressing aberrant formation of interstitial telomeric sequences.
P2860
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P2860
Stable expression of MutLγ in human cells reveals no specific response to mismatched DNA, but distinct recruitment to damage sites.
description
2013 nî lūn-bûn
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2013年の論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年论文
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2013年论文
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2013年论文
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name
Stable expression of MutLγ in ...... t recruitment to damage sites.
@en
Stable expression of MutLγ in ...... t recruitment to damage sites.
@nl
type
label
Stable expression of MutLγ in ...... t recruitment to damage sites.
@en
Stable expression of MutLγ in ...... t recruitment to damage sites.
@nl
prefLabel
Stable expression of MutLγ in ...... t recruitment to damage sites.
@en
Stable expression of MutLγ in ...... t recruitment to damage sites.
@nl
P2093
P2860
P356
P1476
Stable expression of MutLγ in ...... ct recruitment to damage sites
@en
P2093
Christian Mielke
Hans G Drexler
Kurt E J Dittmar
Silke Fähnrich
Yvonne Merkhoffer
P2860
P304
P356
10.1002/JCB.24591
P577
2013-10-01T00:00:00Z