Abortive cell cycle events in the brains of scrapie-infected hamsters with remarkable decreases of PLK3/Cdc25C and increases of PLK1/cyclin B1.
about
Prion-induced neurotoxicity: Possible role for cell cycle activity and DNA damage responseOverexpression of PLK3 Mediates the Degradation of Abnormal Prion Proteins Dependent on Chaperone-Mediated AutophagyActivation of the AMPK-ULK1 pathway plays an important role in autophagy during prion infection.Increases of Galectin-1 and its S-nitrosylated form in the Brain Tissues of Scrapie-Infected Rodent Models and Human Prion Diseases.Aberrant alterations of the expressions and S-nitrosylation of calmodulin and the downstream factors in the brains of the rodents during scrapie infection.Plk1 promotes the migration of human lung adenocarcinoma epithelial cells via STAT3 signaling
P2860
Abortive cell cycle events in the brains of scrapie-infected hamsters with remarkable decreases of PLK3/Cdc25C and increases of PLK1/cyclin B1.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Abortive cell cycle events in ...... d increases of PLK1/cyclin B1.
@en
Abortive cell cycle events in ...... d increases of PLK1/cyclin B1.
@nl
type
label
Abortive cell cycle events in ...... d increases of PLK1/cyclin B1.
@en
Abortive cell cycle events in ...... d increases of PLK1/cyclin B1.
@nl
prefLabel
Abortive cell cycle events in ...... d increases of PLK1/cyclin B1.
@en
Abortive cell cycle events in ...... d increases of PLK1/cyclin B1.
@nl
P2093
P2860
P1476
Abortive cell cycle events in ...... d increases of PLK1/cyclin B1.
@en
P2093
Bao-Yun Zhang
Qi-Xiang Shao
Shao-Bin Wang
P2860
P2888
P304
P356
10.1007/S12035-013-8455-1
P577
2013-04-27T00:00:00Z