There Is a Method to the Madness: Strategies to Study Host Complement Evasion by Lyme Disease and Relapsing Fever Spirochetes

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The changes of gut microbiota after acute myocardial infarction in rats.Polymorphic factor H-binding activity of CspA protects Lyme borreliae from the host complement in feeding ticks to facilitate tick-to-host transmission.

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There Is a Method to the Madness: Strategies to Study Host Complement Evasion by Lyme Disease and Relapsing Fever Spirochetes

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2017 nî lūn-bûn

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There Is a Method to the Madne ...... nd Relapsing Fever Spirochetes

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There Is a Method to the Madne ...... d Relapsing Fever Spirochetes.

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There Is a Method to the Madne ...... nd Relapsing Fever Spirochetes

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There Is a Method to the Madne ...... d Relapsing Fever Spirochetes.

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There Is a Method to the Madne ...... nd Relapsing Fever Spirochetes

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There Is a Method to the Madne ...... d Relapsing Fever Spirochetes.

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There Is a Method to the Madne ...... nd Relapsing Fever Spirochetes

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Ashley L Marcinkiewicz

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Peter Kraiczy

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Yi-Pin Lin

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P2860

Borrelia recurrentis employs a novel multifunctional surface protein with anti-complement, anti-opsonic and invasive potential to escape innate immunity

The emergence of Lyme disease

Differential binding of host complement inhibitor factor H by Borrelia burgdorferi Erp surface proteins: a possible mechanism underlying the expansive host range of Lyme disease spirochetes

Relapsing fever spirochetes Borrelia recurrentis and B. duttonii acquire complement regulators C4b-binding protein and factor H

Human pathogenic Borrelia spielmanii sp. nov. resists complement-mediated killing by direct binding of immune regulators factor H and factor H-like protein 1

Hide and Seek: How Lyme Disease Spirochetes Overcome Complement Attack

Borrelia burgdorferi BBK32 Inhibits the Classical Pathway by Blocking Activation of the C1 Complement Complex

Delineation of Borrelia burgdorferi sensu stricto, Borrelia garinii sp. nov., and group VS461 associated with Lyme borreliosis.

Immune evasion of Borrelia burgdorferi: mapping of a complement-inhibitor factor H-binding site of BbCRASP-3, a novel member of the Erp protein family.

Serum-resistant strains of Borrelia burgdorferi evade complement-mediated killing by expressing a CD59-like complement inhibitory molecule.

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scholarly article

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10.3389/FMICB.2017.00328

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2017-03-02T00:00:00Z

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Lyme disease

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There Is a Method to the Madness: Strategies to Study Host Complement Evasion by Lyme Disease and Relapsing Fever Spirochetes

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P2860

P2860

There Is a Method to the Madness: Strategies to Study Host Complement Evasion by Lyme Disease and Relapsing Fever Spirochetes

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