Silica phagocytosis causes apoptosis and necrosis by different temporal and molecular pathways in alveolar macrophages.
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Cell-Based Therapy for SilicosisSilica particles cause NADPH oxidase-independent ROS generation and transient phagolysosomal leakageRange-finding risk assessment of inhalation exposure to nanodiamonds in a laboratory environmentNanoparticle Uptake: The Phagocyte ProblemMacrophages phagocytose nonopsonized silica particles using a unique microtubule-dependent pathway.Th17 can regulate silica-induced lung inflammation through an IL-1β-dependent mechanismMulti-parametric analysis of cell death pathways using live-cell microscopy.P2X7 receptor modulates inflammatory and functional pulmonary changes induced by silica.IL-10-Producing CD1dhiCD5+ Regulatory B Cells May Play a Critical Role in Modulating Immune Homeostasis in Silicosis Patients.Experimental silicosis does not aggravate collagen-induced arthritis in mice.Estrogenic endocrine-disrupting chemicals modulate the production of inflammatory mediators and cell viability of lipopolysaccharide-stimulated macrophages.Responses of A549 human lung epithelial cells to cristobalite and α-quartz exposures assessed by toxicoproteomics and gene expression analysis.The Effects of Silica Nanoparticles on Apoptosis and Autophagy of Glioblastoma Cell Lines.Single Cell Analysis of Phagocytosis, Phagosome Maturation, Phagolysosomal Leakage, and Cell Death Following Exposure of Macrophages to Silica Particles.Air pollution, oxidative stress, and exacerbation of autoimmune diseases.Association between SNPs at IL-17A and IL-17F and susceptibility to accelerated silicosis.Environmental dust inhalation in the European badger (Meles meles): Systemic distribution of silica-laden macrophages, pathological changes, and association with Mycobacterium bovis infection status.A secretomics analysis reveals major differences in the macrophage responses towards different types of carbon nanotubes.Systematic Overview of Solid Particles and Their Host Responses.Silica nanoparticle-induced oxidative stress and mitochondrial damage is followed by activation of intrinsic apoptosis pathway in glioblastoma cells.4-1BB Signaling Promotes Alveolar Macrophages-Mediated Pro-Fibrotic Responses and Crystalline Silica-Induced Pulmonary Fibrosis in Mice
P2860
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P2860
Silica phagocytosis causes apoptosis and necrosis by different temporal and molecular pathways in alveolar macrophages.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
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2013年论文
@zh-cn
name
Silica phagocytosis causes apo ...... hways in alveolar macrophages.
@en
Silica phagocytosis causes apo ...... hways in alveolar macrophages.
@nl
type
label
Silica phagocytosis causes apo ...... hways in alveolar macrophages.
@en
Silica phagocytosis causes apo ...... hways in alveolar macrophages.
@nl
prefLabel
Silica phagocytosis causes apo ...... hways in alveolar macrophages.
@en
Silica phagocytosis causes apo ...... hways in alveolar macrophages.
@nl
P2860
P1433
P1476
Silica phagocytosis causes apo ...... hways in alveolar macrophages.
@en
P2093
David A Knecht
Gaurav N Joshi
P2860
P2888
P304
P356
10.1007/S10495-012-0798-Y
P577
2013-03-01T00:00:00Z