Increased non-protein bound iron in Down syndrome: contribution to lipid peroxidation and cognitive decline.

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A Comprehensive Diverse '-omics' Approach to Better Understanding the Molecular Pathomechanisms of Down Syndrome.Disturbance of redox homeostasis in Down Syndrome: Role of iron dysmetabolism.

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Increased non-protein bound iron in Down syndrome: contribution to lipid peroxidation and cognitive decline.

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年學術文章

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Increased non-protein bound ir ...... idation and cognitive decline.

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Increased non-protein bound ir ...... idation and cognitive decline.

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type

Item

label

Increased non-protein bound ir ...... idation and cognitive decline.

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Increased non-protein bound ir ...... idation and cognitive decline.

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Increased non-protein bound ir ...... idation and cognitive decline.

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Increased non-protein bound ir ...... idation and cognitive decline.

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Increased non-protein bound ir ...... xidation and cognitive decline

@en

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Caterina Manna

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Cinzia Signorini

http://www.w3.org/2001/XMLSchema#string

Claudio De Felice

http://www.w3.org/2001/XMLSchema#string

Francesca Trojsi

http://www.w3.org/2001/XMLSchema#string

Gioacchino Tedeschi

http://www.w3.org/2001/XMLSchema#string

Silvia Leoncini

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P2860

Alzheimer disease and Down syndrome: factors in pathogenesis

Towards a unifying, systems biology understanding of large-scale cellular death and destruction caused by poorly liganded iron: Parkinson's, Huntington's, Alzheimer's, prions, bactericides, chemical toxicology and others as examples

Chromosome 21 and down syndrome: from genomics to pathophysiology

Novel 5' untranslated region directed blockers of iron-regulatory protein-1 dependent amyloid precursor protein translation: implications for down syndrome and Alzheimer's disease

Serum 8,12-iso-iPF2α-VI isoprostane marker of oxidative damage and cognition deficits in children with konzo

Superoxide dismutase SOD1, encoded on chromosome 21, but not SOD2 is overexpressed in brains of patients with Down syndrome.

Iron chelation in the treatment of neurodegenerative diseases.

The isoprostanes--25 years later

Memory decline in Down syndrome and its relationship to iPF2alpha, a urinary marker of oxidative stress

Challenges associated with metal chelation therapy in Alzheimer's disease

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scholarly article

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10.1080/10715762.2016.1253833

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http://www.w3.org/2001/XMLSchema#string

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Lucia Ciccoli

Arbace Officioso

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2016-11-23T00:00:00Z

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27785947

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Increased non-protein bound iron in Down syndrome: contribution to lipid peroxidation and cognitive decline.

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Increased non-protein bound iron in Down syndrome: contribution to lipid peroxidation and cognitive decline.

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P1433

P1476

P2093

P2860

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P433

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P356

P1433

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P2093

P2860

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P698