Complement-independent blistering mechanisms in bullous pemphigoid.

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Variation of the epidermal expression of glucocorticoid receptor-beta as potential predictive marker of bullous pemphigoid outcome.New insights into pemphigoid diseases.

P2860

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P2860

Complement-independent blistering mechanisms in bullous pemphigoid.

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http://www.wikidata.org/entity/Q39248163

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2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh

2017年學術文章

@zh-hant

name

Complement-independent blistering mechanisms in bullous pemphigoid.

@en

Complement-independent blistering mechanisms in bullous pemphigoid.

@nl

type

Item

label

Complement-independent blistering mechanisms in bullous pemphigoid.

@en

Complement-independent blistering mechanisms in bullous pemphigoid.

@nl

prefLabel

Complement-independent blistering mechanisms in bullous pemphigoid.

@en

Complement-independent blistering mechanisms in bullous pemphigoid.

@nl

P1476

Complement-independent blistering mechanisms in bullous pemphigoid.

@en

P2093

Hideyuki Ujiie

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P2860

The coupling of alpha6beta4 integrin to Ras-MAP kinase pathways mediated by Shc controls keratinocyte proliferation

Cloning and primary structural analysis of the bullous pemphigoid autoantigen BP180

Human antibody-Fc receptor interactions illuminated by crystal structures

Isolation of a human epidermal cDNA corresponding to the 180-kD autoantigen recognized by bullous pemphigoid and herpes gestationis sera. Immunolocalization of this protein to the hemidesmosome.

A passive transfer model of the organ-specific autoimmune disease, bullous pemphigoid, using antibodies generated against the hemidesmosomal antigen, BP180.

Partial cDNA cloning of the 230-kD mouse bullous pemphigoid antigen by use of a human monoclonal anti-basement membrane zone antibody.

The role of complement in experimental bullous pemphigoid.

Inherited epidermolysis bullosa: updated recommendations on diagnosis and classification.

The C5a receptor on mast cells is critical for the autoimmune skin-blistering disease bullous pemphigoid.

Structural features of human immunoglobulin G that determine isotype-specific differences in complement activation.

P304

1235-1239

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P31

scholarly article

P356

10.1111/EXD.13367

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P433

http://www.w3.org/2001/XMLSchema#string

P478

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P50

Hiroaki Iwata

P577

2017-04-18T00:00:00Z

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P698

28418613

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Complement-independent blistering mechanisms in bullous pemphigoid.

about

P2860

P2860

Complement-independent blistering mechanisms in bullous pemphigoid.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P698

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P698