Comparison of non-crystalline silica nanoparticles in IL-1β release from macrophages. - Wikidata - wikimedia - TriplyDB

Comparison of non-crystalline silica nanoparticles in IL-1β release from macrophages.

Comparison of non-crystalline silica nanoparticles in IL-1β release from macrophages.

http://www.wikidata.org/entity/Q39299592

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Sintered indium-tin oxide particles induce pro-inflammatory responses in vitro, in part through inflammasome activation The complex cascade of cellular events governing inflammasome activation and IL-1β processing in response to inhaled particles Silica particles cause NADPH oxidase-independent ROS generation and transient phagolysosomal leakage The NLRP3 inflammasome in pathogenic particle and fibre-associated lung inflammation and diseases Cytokines as biomarkers of nanoparticle immunotoxicity The alarmin IL-1α is a master cytokine in acute lung inflammation induced by silica micro- and nanoparticles Exposure to nickel oxide nanoparticles induces pulmonary inflammation through NLRP3 inflammasome activation in rats Amorphous silica nanoparticles impair vascular homeostasis and induce systemic inflammation Why does the hemolytic activity of silica predict its pro-inflammatory activity?Inflammasome activation in airway epithelial cells after multi-walled carbon nanotube exposure mediates a profibrotic response in lung fibroblasts Intracellular signal modulation by nanomaterials Activation of Proinflammatory Responses in Cells of the Airway Mucosa by Particulate Matter: Oxidant- and Non-Oxidant-Mediated Triggering Mechanisms Differential activation of RAW 264.7 macrophages by size-segregated crystalline silica Revisiting the paradigm of silica pathogenicity with synthetic quartz crystals: the role of crystallinity and surface disorder Reduction of Acute Inflammatory Effects of Fumed Silica Nanoparticles in the Lung by Adjusting Silanol Display through Calcination and Metal Doping Toxic effect of silica nanoparticles on endothelial cells through DNA damage response via Chk1-dependent G2/M checkpoint Crucial Role of Lateral Size for Graphene Oxide in Activating Macrophages and Stimulating Pro-inflammatory Responses in Cells and Animals Effect of silica particle size on macrophage inflammatory responses.Artefactual nanoparticle activation of the inflammasome platform: in vitro evidence with a nano-formed calcium phosphate.Potential of nanoparticles for allergen-specific immunotherapy - use of silica nanoparticles as vaccination platform.Integrative nanomedicine: treating cancer with nanoscale natural products.Role of caspase-1 in nuclear translocation of IL-37, release of the cytokine, and IL-37 inhibition of innate immune responses.Effect of TiO₂ Nanoparticles on Inflammasome-Mediated Airway Inflammation and Responsiveness.The effects of engineered nanoparticles on pulmonary immune homeostasis.Particulate matter-mediated release of long pentraxin 3 (PTX3) and vascular endothelial growth factor (VEGF) in vitro: Limited importance of endotoxin and organic content.Carbon Nanotubes Disrupt Iron Homeostasis and Induce Anemia of Inflammation through Inflammatory Pathway as a Secondary Effect Distant to Their Portal-of-Entry.Applicability of avidin protein coated mesoporous silica nanoparticles as drug carriers in the lung.Proinflammatory Effects of Pyrogenic and Precipitated Amorphous Silica Nanoparticles in Innate Immunity Cells.Extracellular cathepsin S and intracellular caspase 1 activation are surrogate biomarkers of particulate-induced lysosomal disruption in macrophages The role of Rho-kinases in IL-1β release through phagocytosis of fibrous particles in human monocytes.Nano-sized and micro-sized polystyrene particles affect phagocyte function.Release of IL-1 β triggered by Milan summer PM10: molecular pathways involved in the cytokine release.Management of traveller's diarrhoea with a combination of sodium butyrate, organic acids, and A-300 silicon dioxide Contamination of nanoparticles by endotoxin: evaluation of different test methods.Nonlinear effects of nanoparticles: biological variability from hormetic doses, small particle sizes, and dynamic adaptive interactions.Titanium ions form particles that activate and execute interleukin-1β release from lipopolysaccharide-primed macrophages.Caspase-1 inhibition attenuates activation of BV2 microglia induced by LPS-treated RAW264.7 macrophages.Combined Action of Human Commensal Bacteria and Amorphous Silica Nanoparticles on the Viability and Immune Responses of Dendritic Cells.Structure Activity Relationships of Engineered Nanomaterials in inducing NLRP3 Inflammasome Activation and Chronic Lung Fibrosis.Dioscin Exerts Protective Effects Against Crystalline Silica-induced Pulmonary Fibrosis in Mice.

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Comparison of non-crystalline silica nanoparticles in IL-1β release from macrophages.

http://www.wikidata.org/entity/Q39299592

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2012 nî lūn-bûn

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年學術文章

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2012年學術文章

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name

Comparison of non-crystalline silica nanoparticles in IL-1β release from macrophages.

@en

Comparison of non-crystalline silica nanoparticles in IL-1β release from macrophages.

@nl

type

label

Comparison of non-crystalline silica nanoparticles in IL-1β release from macrophages.

@en

Comparison of non-crystalline silica nanoparticles in IL-1β release from macrophages.

@nl

prefLabel

Comparison of non-crystalline silica nanoparticles in IL-1β release from macrophages.

@en

Comparison of non-crystalline silica nanoparticles in IL-1β release from macrophages.

@nl

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Particle and Fibre Toxicology

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Comparison of non-crystalline silica nanoparticles in IL-1β release from macrophages.

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Bernd Friede

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Magne Refsnes

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Marit Låg

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Per E Schwarze

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Tonje Skuland

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Wiggo J Sandberg

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P2860

Mechanisms in the Pathogenesis of Asbestosis and Silicosis

The inflammasomes

The inflammasome: a molecular platform triggering activation of inflammatory caspases and processing of proIL-beta

Nanoparticles activate the NLR pyrin domain containing 3 (Nlrp3) inflammasome and cause pulmonary inflammation through release of IL-1α and IL-1β

Silica crystals and aluminum salts activate the NALP3 inflammasome through phagosomal destabilization

The Nalp3 inflammasome is essential for the development of silicosis

Innate immune activation through Nalp3 inflammasome sensing of asbestos and silica

Is silicosis required for silica-associated lung cancer?

Gout-associated uric acid crystals activate the NALP3 inflammasome

Inflammatory caspases and inflammasomes: master switches of inflammation

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10.1186/1743-8977-9-32

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9

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Jørn Andreas Holme

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2012-08-10T00:00:00Z

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230654707

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