Autophagy is a cell self-protective mechanism against arsenic-induced cell transformation. - Wikidata - wikimedia - TriplyDB

Autophagy is a cell self-protective mechanism against arsenic-induced cell transformation.

Autophagy is a cell self-protective mechanism against arsenic-induced cell transformation.

http://www.wikidata.org/entity/Q39301296

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Gene expression profile of human lung epithelial cells chronically exposed to single-walled carbon nanotubes Carbon nanotubes induce apoptosis resistance of human lung epithelial cells through FLICE-inhibitory protein Oxidative stress-induced autophagy: role in pulmonary toxicity Arsenite-induced pseudo-hypoxia results in loss of anchorage-dependent growth in BEAS-2B pulmonary epithelial cells Arsenic exposure induces the Warburg effect in cultured human cells Progress and prospects of reactive oxygen species in metal carcinogenesis Autophagy inhibition by sustained overproduction of IL6 contributes to arsenic carcinogenesis The impact of oil spill to lung health--Insights from an RNA-seq study of human airway epithelial cells Culture conditions profoundly impact phenotype in BEAS-2B, a human pulmonary epithelial model Evaluation of tumorigenic potential of CeO2 and Fe2O3 engineered nanoparticles by a human cell in vitro screening model M2 polarization of macrophages facilitates arsenic-induced cell transformation of lung epithelial cells Arsenic induces sustained impairment of skeletal muscle and muscle progenitor cell ultrastructure and bioenergetics.Role of Nrf2 and Autophagy in Acute Lung Injury Autophagy as a double-edged sword in pulmonary epithelial injury: a review and perspective.Arsenic Compromises Both p97 and Proteasome Functions.Deficiencies in mitochondrial dynamics sensitize Caenorhabditis elegans to arsenite and other mitochondrial toxicants by reducing mitochondrial adaptability.Hypoxia induces autophagy in cardiomyocytes via a hypoxia-inducible factor 1-dependent mechanism.The p-eIF2α/ATF4 pathway links endoplasmic reticulum stress to autophagy following the production of reactive oxygen species in mouse spermatocyte-derived cells exposed to dibutyl phthalate.From the Cover: Arsenite Uncouples Mitochondrial Respiration and Induces a Warburg-like Effect in Caenorhabditis elegans.STAT3 in arsenic lung carcinogenicity.Arsenic-induced suppression of kidney cell proliferation and the transcriptional coregulator MAML1.Alterations of miRNAs and Their Potential Roles in Arsenite-Induced Transformation of Human Bronchial Epithelial Cells.Arsenite downregulates H3K4 trimethylation and H3K9 dimethylation during transformation of human bronchial epithelial cells.Autophagy upregulation as a possible mechanism of arsenic induced diabetes

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Autophagy is a cell self-protective mechanism against arsenic-induced cell transformation.

http://www.wikidata.org/entity/Q39301296

description

2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年學術文章

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2012年學術文章

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2012年學術文章

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name

Autophagy is a cell self-prote ...... c-induced cell transformation.

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Autophagy is a cell self-prote ...... c-induced cell transformation.

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type

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Autophagy is a cell self-prote ...... c-induced cell transformation.

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Autophagy is a cell self-prote ...... c-induced cell transformation.

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Autophagy is a cell self-prote ...... c-induced cell transformation.

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Autophagy is a cell self-prote ...... c-induced cell transformation.

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Toxicological Sciences

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Autophagy is a cell self-prote ...... c-induced cell transformation.

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P2093

Gang Chen

http://www.w3.org/2001/XMLSchema#string

Jacqueline A Frank

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Jia Luo

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Kimberley A Bower

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Mei Xu

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Mingjun Liao

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Tao Zhang

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P2860

Molecular mechanisms of arsenic carcinogenesis

Phosphorylation and regulation of Akt/PKB by the rictor-mTOR complex

Functional and physical interaction between Bcl-X(L) and a BH3-like domain in Beclin-1

Beclin 1, an autophagy gene essential for early embryonic development, is a haploinsufficient tumor suppressor

Mammalian autophagy: core molecular machinery and signaling regulation

Promotion of tumorigenesis by heterozygous disruption of the beclin 1 autophagy gene

Autophagy facilitates the development of breast cancer resistance to the anti-HER2 monoclonal antibody trastuzumab

Induction of autophagy and inhibition of tumorigenesis by beclin 1

NADPH oxidase activation is required in reactive oxygen species generation and cell transformation induced by hexavalent chromium

Reduced basal autophagy and impaired mitochondrial dynamics due to loss of Parkinson's disease-associated protein DJ-1

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