Nitric oxide selectively depletes macrophages in atherosclerotic plaques via induction of endoplasmic reticulum stress. - Wikidata - wikimedia - TriplyDB

Nitric oxide selectively depletes macrophages in atherosclerotic plaques via induction of endoplasmic reticulum stress.

Nitric oxide selectively depletes macrophages in atherosclerotic plaques via induction of endoplasmic reticulum stress.

http://www.wikidata.org/entity/Q39321586

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Involvement of the IRE1α-XBP1 pathway and XBP1s-dependent transcriptional reprogramming in metabolic diseases Mouse aorta-derived mesenchymal progenitor cells contribute to and enhance the immune response of macrophage cells under inflammatory conditions Acute exposure to methamphetamine alters TLR9-mediated cytokine expression in human macrophage.Overexpression of X-Box Binding Protein 1 (XBP1) Correlates to Poor Prognosis and Up-Regulation of PI3K/mTOR in Human Osteosarcoma.Pharmacological modulation of cell death in atherosclerosis: a promising approach towards plaque stabilization?Nitric oxide: a regulator of eukaryotic initiation factor 2 kinases Therapeutic strategies to deplete macrophages in atherosclerotic plaques.Tunicamycin inhibits PDGF-BB-induced proliferation and migration of vascular smooth muscle cells through induction of HO-1.

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P2860

Nitric oxide selectively depletes macrophages in atherosclerotic plaques via induction of endoplasmic reticulum stress.

http://www.wikidata.org/entity/Q39321586

description

2007 nî lūn-bûn

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2007年の論文

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年學術文章

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2007年學術文章

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2007年學術文章

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name

Nitric oxide selectively deple ...... endoplasmic reticulum stress.

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Nitric oxide selectively deple ...... endoplasmic reticulum stress.

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type

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Nitric oxide selectively deple ...... endoplasmic reticulum stress.

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Nitric oxide selectively deple ...... endoplasmic reticulum stress.

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British Journal of Pharmacology

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Nitric oxide selectively deple ...... endoplasmic reticulum stress.

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P2093

A G Herman

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G R Y De Meyer

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V Croons

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W Martinet

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P2860

Roles of CHOP/GADD153 in endoplasmic reticulum stress

Prolonged endoplasmic reticulum stress in hypertrophic and failing heart after aortic constriction: possible contribution of endoplasmic reticulum stress to cardiac myocyte apoptosis

Gadd153 sensitizes cells to endoplasmic reticulum stress by down-regulating Bcl2 and perturbing the cellular redox state

Inflammation in atherosclerosis

A trip to the ER: coping with stress

Pathophysiology and clinical significance of atherosclerotic plaque rupture.

Strategies to achieve coronary arterial plaque stabilization.

Elevated levels of oxidative DNA damage and DNA repair enzymes in human atherosclerotic plaques.

Consequences of cellular cholesterol accumulation: basic concepts and physiological implications

The endoplasmic reticulum: a multifunctional signaling organelle.

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493-500

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10.1038/SJ.BJP.0707426

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4

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152

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Jean-Pierre Timmermans

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2007-08-13T00:00:00Z

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6138466

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17700714

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atherosclerosis

endoplasmic reticulum

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2050816

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