Human papillomaviruses recruit cellular DNA repair and homologous recombination factors to viral replication centers. - Wikidata - wikimedia - TriplyDB

Human papillomaviruses recruit cellular DNA repair and homologous recombination factors to viral replication centers.

Human papillomaviruses recruit cellular DNA repair and homologous recombination factors to viral replication centers.

http://www.wikidata.org/entity/Q39323899

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Inactivation of p53 rescues the maintenance of high risk HPV DNA genomes deficient in expression of E6 Modulation of DNA damage and repair pathways by human tumour viruses Mechanisms of viral mutation Molecular mechanisms of HPV mediated neoplastic progression The Role of the DNA Damage Response throughout the Papillomavirus Life Cycle The acetyltransferase Tip60 is a critical regulator of the differentiation-dependent amplification of human papillomaviruses.Papillomavirus genomes associate with BRD4 to replicate at fragile sites in the host genome.Reverse-phase protein array profiling of oropharyngeal cancer and significance of PIK3CA mutations in HPV-associated head and neck cancer.Human papillomavirus type 16 E6* induces oxidative stress and DNA damage.Productive replication of human papillomavirus 31 requires DNA repair factor Nbs1.Merkel cell polyomavirus: molecular insights into the most recently discovered human tumour virus.CD66+ cells in cervical precancers are partially differentiated progenitors with neoplastic traits Recruitment of Brd4 to the human papillomavirus type 16 DNA replication complex is essential for replication of viral DNA.Papillomaviruses use recombination-dependent replication to vegetatively amplify their genomes in differentiated cells DNA damage repair genes controlling human papillomavirus (HPV) episome levels under conditions of stability and extreme instability Brd4 is displaced from HPV replication factories as they expand and amplify viral DNA Proteomic analysis and identification of cellular interactors of the giant ubiquitin ligase HERC2.Recent Insights into the Control of Human Papillomavirus (HPV) Genome Stability, Loss, and Degradation.Activation of NF-κB by human papillomavirus 16 E1 limits E1-dependent viral replication through degradation of E1.SV40 utilizes ATM kinase activity to prevent non-homologous end joining of broken viral DNA replication products Human papillomaviruses activate and recruit SMC1 cohesin proteins for the differentiation-dependent life cycle through association with CTCF insulators The Cell Cycle Timing of Human Papillomavirus DNA Replication.The Deacetylase Sirtuin 1 Regulates Human Papillomavirus Replication by Modulating Histone Acetylation and Recruitment of DNA Damage Factors NBS1 and Rad51 to Viral Genomes.The role of the PI3K/Akt/mTOR signalling pathway in human cancers induced by infection with human papillomaviruses.What's the damage? The impact of pathogens on pathways that maintain host genome integrity Replication of an Autonomous Human Parvovirus in Non-dividing Human Airway Epithelium Is Facilitated through the DNA Damage and Repair Pathways Impact of the DNA Damage Response on Human Papillomavirus Chromatin.The Replicative Consequences of Papillomavirus E2 Protein Binding to the Origin Replication Factor ORC2 Human papillomaviruses: shared and distinct pathways for pathogenesis Engagement of the ATR-dependent DNA damage response at the human papillomavirus 18 replication centers during the initial amplification.SMC1-mediated intra-S-phase arrest facilitates bocavirus DNA replication.Nucleotide Excision Repair Gene ERCC2 and ERCC5 Variants Increase Risk of Uterine Cervical Cancer.Current understanding of the role of the Brd4 protein in the papillomavirus lifecycle.DNA Damage Reduces the Quality, but Not the Quantity of Human Papillomavirus 16 E1 and E2 DNA Replication Merkel cell polyomavirus large T antigen disrupts host genomic integrity and inhibits cellular proliferation.The papillomavirus E2 proteins Recombination-dependent oligomerization of human papillomavirus genomes upon transient DNA replication.The E1 proteins.Genome-wide analysis of HPV integration in human cancers reveals recurrent, focal genomic instability Regulation of the life cycle of HPVs by differentiation and the DNA damage response

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P2860

Human papillomaviruses recruit cellular DNA repair and homologous recombination factors to viral replication centers.

http://www.wikidata.org/entity/Q39323899

description

2012 nî lūn-bûn

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Journal of Virology

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Cary A Moody

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Kavi P Mehta

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Kenric A Gillespie

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Laimonis A Laimins

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P2860

The mechanism of double-strand DNA break repair by the nonhomologous DNA end-joining pathway

ATM activation by DNA double-strand breaks through the Mre11-Rad50-Nbs1 complex

ATM and ATR substrate analysis reveals extensive protein networks responsive to DNA damage

Human Rif1, ortholog of a yeast telomeric protein, is regulated by ATM and 53BP1 and functions in the S-phase checkpoint

Multiple roles of ATM in monitoring and maintaining DNA integrity

The DNA Damage Response: Making It Safe to Play with Knives

The DNA damage response: ten years after

Mitotic homologous recombination maintains genomic stability and suppresses tumorigenesis

H2AX: the histone guardian of the genome

Replication protein A phosphorylation and the cellular response to DNA damage

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9520-9526

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10.1128/JVI.00247-12

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17

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2012-06-27T00:00:00Z

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22740399

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3416172

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