Chronic exposure to laminar shear stress induces Kruppel-like factor 2 in glomerular endothelial cells and modulates interactions with co-cultured podocytes.

Chronic exposure to laminar shear stress induces Kruppel-like factor 2 in glomerular endothelial cells and modulates interactions with co-cultured podocytes.

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http://www.wikidata.org/entity/Q39334237
Comprehensive analysis of glomerular mRNA expression of pro- and antithrombotic genes in atypical haemolytic-uremic syndrome (aHUS).Glomerular endothelial cell injury and damage precedes that of podocytes in adriamycin-induced nephropathy.Reduced Krüppel-like factor 2 expression may aggravate the endothelial injury of diabetic nephropathy.Glomerular endothelial cell injury and cross talk in diabetic kidney diseasePodocyte injury-driven intracapillary plasminogen activator inhibitor type 1 accelerates podocyte loss via uPAR-mediated β1-integrin endocytosis.Loss of Krüppel-like factor 6 cripples podocyte mitochondrial function.The decrease in histone methyltransferase EZH2 in response to fluid shear stress alters endothelial gene expression and promotes quiescence.Endothelial-podocyte crosstalk: the missing link between endothelial dysfunction and albuminuria in diabetes.Epigenetic markers of renal function in african americans.The role of ERK5 in endothelial cell function.Exosomes from high glucose-treated glomerular endothelial cells trigger the epithelial-mesenchymal transition and dysfunction of podocytesThe critical role of Krüppel-like factors in kidney disease.Reduced Krüppel-Like Factor 2 Aggravates Glomerular Endothelial Cell Injury and Kidney Disease in Mice with Unilateral Nephrectomy.Mechanisms of Hypothermic Machine Perfusion to Decrease Donation After Cardiac Death Graft Inflammation: Through the Pathway of Upregulating Expression of KLF2 and Inhibiting TGF-β Signaling.One or 4 h of "in-house" reconditioning by machine perfusion after cold storage improve reperfusion parameters in porcine kidneys.Identification of Basic Fibroblast Growth Factor as the Dominant Protector of Laminar Shear Medium from the Modified Shear Device in Tumor Necrosis Factor-α Induced Endothelial Dysfunction.Inhibition of Rho-associated kinase relieves C5a-induced proteinuria in murine nephrotic syndrome.
P2860

Chronic exposure to laminar shear stress induces Kruppel-like factor 2 in glomerular endothelial cells and modulates interactions with co-cultured podocytes.

Item
http://www.wikidata.org/entity/Q39334237
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh
2012年學術文章
@zh-hant
name
Chronic exposure to laminar sh ...... ns with co-cultured podocytes.
@en
Chronic exposure to laminar sh ...... ns with co-cultured podocytes.
@nl
type
label
Chronic exposure to laminar sh ...... ns with co-cultured podocytes.
@en
Chronic exposure to laminar sh ...... ns with co-cultured podocytes.
@nl
prefLabel
Chronic exposure to laminar sh ...... ns with co-cultured podocytes.
@en
Chronic exposure to laminar sh ...... ns with co-cultured podocytes.
@nl
P1433
P1476
P2093
P304
P31
P356
P433
P478
P50
P577
P5875
P698
P921
P356
P1433
P1476
Chronic exposure to laminar sh ...... ns with co-cultured podocytes.
@en
P2093
P304
P31
P356
10.1016/J.BIOCEL.2012.05.020
P433
P478
P50
P577
P5875
P698
P921