Repression of interleukin-5 transcription by the glucocorticoid receptor targets GATA3 signaling and involves histone deacetylase recruitment.
about
Suppression of GATA-3 nuclear import and phosphorylation: a novel mechanism of corticosteroid action in allergic diseaseHistone deacetylase 1 (HDAC1) participates in the down-regulation of corticotropin releasing hormone gene (crh) expressionMinireview: latest perspectives on antiinflammatory actions of glucocorticoids.SATB1 dictates expression of multiple genes including IL-5 involved in human T helper cell differentiation.Antiinflammatory glucocorticoid receptor ligand with reduced side effects exhibits an altered protein-protein interaction profile.TH17 cells mediate steroid-resistant airway inflammation and airway hyperresponsiveness in mice.Mechanisms involved in glucocorticoid induction of pituitary GH expression during embryonic developmentGATA3 up-regulation associated with surface expression of CD294/CRTH2: a unique feature of human Th cells.Glucocorticoid receptor mediated suppression of natural killer cell activity: identification of associated deacetylase and corepressor molecules.New drugs targeting Th2 lymphocytes in asthma.Donor-derived exosomes induce specific regulatory T cells to suppress immune inflammation in the allograft heartMolecular understanding of cytokine-steroid hormone dialogue: implications for human diseases.Interleukin-13 signaling and its role in asthmaRestraint stress modulates virus specific adaptive immunity during acute Theiler's virus infectionHistone deacetylases and the immunological network: implications in cancer and inflammation.Modulation of antigen-presenting cells by HDAC inhibitors: implications in autoimmunity and cancer.Epigenetics in asthma and other inflammatory lung diseases.Linking GATA-3 and interleukin-13: implications in asthma.Agonist-specific Protein Interactomes of Glucocorticoid and Androgen Receptor as Revealed by Proximity Mapping.The Interactome of the Glucocorticoid Receptor and Its Influence on the Actions of Glucocorticoids in Combatting Inflammatory and Infectious Diseases.Repression of TNF-alpha-induced IL-8 expression by the glucocorticoid receptor-beta involves inhibition of histone H4 acetylation.Recruitment of histone deacetylase 4 by transcription factors represses interleukin-5 transcription.
P2860
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P2860
Repression of interleukin-5 transcription by the glucocorticoid receptor targets GATA3 signaling and involves histone deacetylase recruitment.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Repression of interleukin-5 tr ...... stone deacetylase recruitment.
@en
Repression of interleukin-5 tr ...... stone deacetylase recruitment.
@nl
type
label
Repression of interleukin-5 tr ...... stone deacetylase recruitment.
@en
Repression of interleukin-5 tr ...... stone deacetylase recruitment.
@nl
prefLabel
Repression of interleukin-5 tr ...... stone deacetylase recruitment.
@en
Repression of interleukin-5 tr ...... stone deacetylase recruitment.
@nl
P2093
P2860
P50
P356
P1476
Repression of interleukin-5 tr ...... stone deacetylase recruitment.
@en
P2093
Audrey Kelly
Cecilia Soh
David Richards
Holly Bowen
Jane Gilmour
Philip Smith
P2860
P304
23243-23250
P356
10.1074/JBC.M503659200
P407
P577
2005-04-11T00:00:00Z