MYC directs transcription of MCL1 and eIF4E genes to control sensitivity of gastric cancer cells toward HDAC inhibitors.
about
Dinaciclib potently suppresses MCL-1 and selectively induces the cell death in human iPS cells without affecting the viability of cardiac tissue.Histone deacetylase 1/Sp1/microRNA-200b signaling accounts for maintenance of cancer stem-like cells in human lung adenocarcinomaCDK7 inhibition suppresses super-enhancer-linked oncogenic transcription in MYCN-driven cancer.Wnt modulates MCL1 to control cell survival in triple negative breast cancer.Inhibition of histone deacetylase 4 increases cytotoxicity of docetaxel in gastric cancer cells.Targeting a ribonucleoprotein complex containing the caprin-1 protein and the c-Myc mRNA suppresses tumor growth in mice: an identification of a novel oncotargetThe soy isoflavone equol may increase cancer malignancy via up-regulation of eukaryotic protein synthesis initiation factor eIF4GEfemp1 and p27(Kip1) modulate responsiveness of pancreatic cancer cells towards a dual PI3K/mTOR inhibitor in preclinical models.Galectin-7 is epigenetically-regulated tumor suppressor in gastric cancer.Eukaryotic translation initiation factor 4E as a novel therapeutic target in hematological malignancies and beyond.Biological effects of the Pim kinase inhibitor, SGI-1776, in multiple myeloma.PIM1 kinase regulates cell death, tumor growth and chemotherapy response in triple-negative breast cancer.Role of micro-RNAs in drug resistance of multiple myeloma.MYC and EGR1 synergize to trigger tumor cell death by controlling NOXA and BIM transcription upon treatment with the proteasome inhibitor bortezomib.HDAC1 and HDAC2 integrate the expression of p53 mutants in pancreatic cancer.MicroRNA-561 inhibits gastric cancercell proliferation and invasion by downregulating c-Myc expression.MYC and MCL1 Cooperatively Promote Chemotherapy-Resistant Breast Cancer Stem Cells via Regulation of Mitochondrial Oxidative Phosphorylation.4-Phenybutyric acid promotes gastric cancer cell migration via histone deacetylase inhibition-mediated HER3/HER4 up-regulation.Cisplatin-mediated c-myc overexpression and cytochrome c (cyt c) release result in the up-regulation of the death receptors DR4 and DR5 and the activation of caspase 3 and caspase 9, likely responsible for the TRAIL-sensitizing effect of cisplatin.Genome-wide Analyses on Single Disease Samples for Potential Biomarkers and Biological Features of Molecular Subtypes: A Case Study in Gastric Cancer.
P2860
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P2860
MYC directs transcription of MCL1 and eIF4E genes to control sensitivity of gastric cancer cells toward HDAC inhibitors.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
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2012年學術文章
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name
MYC directs transcription of M ...... cells toward HDAC inhibitors.
@en
MYC directs transcription of M ...... cells toward HDAC inhibitors.
@nl
type
label
MYC directs transcription of M ...... cells toward HDAC inhibitors.
@en
MYC directs transcription of M ...... cells toward HDAC inhibitors.
@nl
prefLabel
MYC directs transcription of M ...... cells toward HDAC inhibitors.
@en
MYC directs transcription of M ...... cells toward HDAC inhibitors.
@nl
P2093
P2860
P50
P356
P1433
P1476
MYC directs transcription of M ...... cells toward HDAC inhibitors.
@en
P2093
Dieter Saur
Günter Schneider
Matthias Wirth
Natasa Stojanovic
Roland M Schmid
Wajana L Labisso
P2860
P304
P356
10.4161/CC.20008
P577
2012-04-15T00:00:00Z