BosR (BB0647) governs virulence expression in Borrelia burgdorferi.
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Analysis of the Borrelia burgdorferi cyclic-di-GMP-binding protein PlzA reveals a role in motility and virulenceBorrelia burgdorferi requires the alternative sigma factor RpoS for dissemination within the vector during tick-to-mammal transmissionStage-specific global alterations in the transcriptomes of Lyme disease spirochetes during tick feeding and following mammalian host adaptation.Multifunctional and Redundant Roles of Borrelia burgdorferi Outer Surface Proteins in Tissue Adhesion, Colonization, and Complement EvasionMicroarray-Based Comparative Genomic and Transcriptome Analysis of Borrelia burgdorferiRole of acetyl-phosphate in activation of the Rrp2-RpoN-RpoS pathway in Borrelia burgdorferiCarbon storage regulator A (CsrA(Bb)) is a repressor of Borrelia burgdorferi flagellin protein FlaB.The cyclic-di-GMP signaling pathway in the Lyme disease spirochete, Borrelia burgdorferiInvestigation of ospC Expression Variation among Borrelia burgdorferi StrainsTranscriptional responses of Leptospira interrogans to host innate immunity: significant changes in metabolism, oxygen tolerance, and outer membraneBosR (BB0647) controls the RpoN-RpoS regulatory pathway and virulence expression in Borrelia burgdorferi by a novel DNA-binding mechanism.The bba64 gene of Borrelia burgdorferi, the Lyme disease agent, is critical for mammalian infection via tick bite transmission.Functional analysis of the Borrelia burgdorferi bba64 gene product in murine infection via tick infestation.Cyclic di-GMP is essential for the survival of the lyme disease spirochete in ticksBorrelia burgdorferi requires glycerol for maximum fitness during the tick phase of the enzootic cycle.HrpA, a DEAH-box RNA helicase, is involved in global gene regulation in the Lyme disease spirochete.Transcriptional response of Leptospira interrogans to iron limitation and characterization of a PerR homologWho is the BosR around here anyway?CsrA modulates levels of lipoproteins and key regulators of gene expression critical for pathogenic mechanisms of Borrelia burgdorferi.RpoS regulates essential virulence factors remaining to be identified in Borrelia burgdorferi.BB0844, an RpoS-regulated protein, is dispensable for Borrelia burgdorferi infectivity and maintenance in the mouse-tick infectious cycle.Global repression of host-associated genes of the Lyme disease spirochete through post-transcriptional modulation of the alternative sigma factor RpoSNitrosative damage to free and zinc-bound cysteine thiols underlies nitric oxide toxicity in wild-type Borrelia burgdorferi.Analysis of the HD-GYP domain cyclic dimeric GMP phosphodiesterase reveals a role in motility and the enzootic life cycle of Borrelia burgdorferi.Oligopeptide permease A5 modulates vertebrate host-specific adaptation of Borrelia burgdorferi.Synthesis of RpoS is dependent on a putative enhancer binding protein Rrp2 in Borrelia burgdorferi.Borrelia burgdorferi linear plasmid 38 is dispensable for completion of the mouse-tick infectious cycle.BosR functions as a repressor of the ospAB operon in Borrelia burgdorferi.Characterization of the RelBbu Regulon in Borrelia burgdorferi Reveals Modulation of Glycerol Metabolism by (p)ppGpp.The coenzyme A disulphide reductase of Borrelia burgdorferi is important for rapid growth throughout the enzootic cycle and essential for infection of the mammalian hostThe Borrelia burgdorferi RelA/SpoT Homolog and Stringent Response Regulate Survival in the Tick Vector and Global Gene Expression during StarvationActivation of the RpoN-RpoS regulatory pathway during the enzootic life cycle of Borrelia burgdorferi.In Vivo Imaging Demonstrates That Borrelia burgdorferi ospC Is Uniquely Expressed Temporally and Spatially throughout Experimental InfectionA high-throughput genetic screen identifies previously uncharacterized Borrelia burgdorferi genes important for resistance against reactive oxygen and nitrogen species.Changes in bacterial growth rate govern expression of the Borrelia burgdorferi OspC and Erp infection-associated surface proteinsBorrelia burgdorferi and tick proteins supporting pathogen persistence in the vector.Virulence of the Lyme disease spirochete before and after the tick bloodmeal: a quantitative assessment.The Fur homologue BosR requires Arg39 to activate rpoS transcription in Borrelia burgdorferi and thereby direct spirochaete infection in mice.Study of the response regulator Rrp1 reveals its regulatory role in chitobiose utilization and virulence of Borrelia burgdorferi.Insight into the Dual Functions of Bacterial Enhancer-Binding Protein Rrp2 of Borrelia burgdorferi
P2860
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P2860
BosR (BB0647) governs virulence expression in Borrelia burgdorferi.
description
2009 nî lūn-bûn
@nan
2009年の論文
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2009年学术文章
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2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
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2009年學術文章
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2009年學術文章
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name
BosR (BB0647) governs virulence expression in Borrelia burgdorferi.
@en
BosR
@nl
type
label
BosR (BB0647) governs virulence expression in Borrelia burgdorferi.
@en
BosR
@nl
prefLabel
BosR (BB0647) governs virulence expression in Borrelia burgdorferi.
@en
BosR
@nl
P2093
P2860
P1476
BosR (BB0647) governs virulence expression in Borrelia burgdorferi
@en
P2093
Martin Goldberg
Michael V Norgard
Shayma Haq
Toru Kariu
Zhiming Ouyang
P2860
P304
P356
10.1111/J.1365-2958.2009.06945.X
P407
P50
P577
2009-11-02T00:00:00Z