The Ability of Extracellular Vesicles to Induce a Pro-Inflammatory Host Response.

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From Inflammasome to Exosome-Does Extracellular Vesicle Secretion Constitute an Inflammasome-Dependent Immune Response?

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Q57492013-11A0553C-D828-43B2-A597-ACC7E5BD99F7

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The Ability of Extracellular Vesicles to Induce a Pro-Inflammatory Host Response.

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The Ability of Extracellular Vesicles to Induce a Pro-Inflammatory Host Response.

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The Ability of Extracellular Vesicles to Induce a Pro-Inflammatory Host Response.

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The Ability of Extracellular Vesicles to Induce a Pro-Inflammatory Host Response.

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The Ability of Extracellular Vesicles to Induce a Pro-Inflammatory Host Response.

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The Ability of Extracellular Vesicles to Induce a Pro-Inflammatory Host Response.

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The Ability of Extracellular Vesicles to Induce a Pro-Inflammatory Host Response.

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P1476

The Ability of Extracellular Vesicles to Induce a Pro-Inflammatory Host Response.

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Maike E van Hezel

http://www.w3.org/2001/XMLSchema#string

Rienk Nieuwland

http://www.w3.org/2001/XMLSchema#string

Robin van Bruggen

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Creative Commons Attribution

P2860

Platelets amplify inflammation in arthritis via collagen-dependent microparticle production

Synovial microparticles from arthritic patients modulate chemokine and cytokine release by synoviocytes

Classification, functions, and clinical relevance of extracellular vesicles

Nonclassical IL-1 beta secretion stimulated by P2X7 receptors is dependent on inflammasome activation and correlated with exosome release in murine macrophages

Severe secondary deficiency of von Willebrand factor-cleaving protease (ADAMTS13) in patients with sepsis-induced disseminated intravascular coagulation: its correlation with development of renal failure.

Monocyte derived microvesicles deliver a cell death message via encapsulated caspase-1.

Microparticles from stored red blood cells activate neutrophils and cause lung injury after hemorrhage and resuscitation.

Activation of macrophages by P2X7-induced microvesicles from myeloid cells is mediated by phospholipids and is partially dependent on TLR4.

Cellular microparticle and thrombogram phenotypes in the Prospective Observational Multicenter Major Trauma Transfusion (PROMMTT) study: correlation with coagulopathy.

Microparticles mediate hepatic ischemia-reperfusion injury and are the targets of Diannexin (ASP8597)

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scholarly article

review article

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10.3390/IJMS18061285

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English

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http://www.w3.org/2001/XMLSchema#string

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Nicole P Juffermans

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2017-06-16T00:00:00Z

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28621719

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Achyolli

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5486107

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The Ability of Extracellular Vesicles to Induce a Pro-Inflammatory Host Response.

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P2860

P2860

The Ability of Extracellular Vesicles to Induce a Pro-Inflammatory Host Response.

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P1433

P1476

P2093

P275

P2860

P31

P356

P407

P433

P478

P50

P577

P698

P921

P932

P356

P1433

P1476

P2093

P275

P2860

P31

P356

P407

P433

P478

P50

P577

P698

P921

P932