Relief of oxidative stress by ascorbic acid delays cellular senescence of normal human and Werner syndrome fibroblast cells.
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Senescence in Human Mesenchymal Stem Cells: Functional Changes and Implications in Stem Cell-Based TherapyOxidative stress-induced telomeric erosion as a mechanism underlying airborne particulate matter-related cardiovascular diseaseAging and immune function: molecular mechanisms to interventionsMultivitamin use and telomere length in womenUsage of Human Mesenchymal Stem Cells in Cell-based Therapy: Advantages and DisadvantagesCellular phenotype-dependent and -independent effects of vitamin C on the renewal and gene expression of mouse embryonic fibroblastsVitamin C restores healthy aging in a mouse model for Werner syndromeUltraviolet radiation, aging and the skin: prevention of damage by topical cAMP manipulationReduction of mitochondrial H2O2 by overexpressing peroxiredoxin 3 improves glucose tolerance in miceThe SLC23 family of ascorbate transporters: ensuring that you get and keep your daily dose of vitamin CNAC selectively inhibit cancer telomerase activity: A higher redox homeostasis threshold exists in cancer cells.WRN Mutation Update: Mutation Spectrum, Patient Registries, and Translational ProspectsEffects of bioactive compounds on senescence and components of senescence associated secretory phenotypes in vitro.Cellular lifespan and senescence: a complex balance between multiple cellular pathways.Ascorbate prevents cell death from prolonged exposure to glutamate in an in vitro model of human dopaminergic neurons.Senescence-induced increases in intracellular oxidative stress and enhancement of the need for ascorbic acid in human fibroblasts.Different involvement of radical species in irradiated and bystander cells.Ascorbic acid extends replicative life span of human embryonic fibroblast by reducing DNA and mitochondrial damages.Electrochemical direct determination of catecholamines for the early detection of neurodegenerative diseases.Persistent amplification of DNA damage signal involved in replicative senescence of normal human diploid fibroblastsActivation of p38 MAP kinase and stress signalling in fibroblasts from the progeroid Rothmund-Thomson syndrome.Ascorbic Acid Attenuates Senescence of Human Osteoarthritic Osteoblasts.Protective roles of ascorbic acid in oxidative stress induced by depletion of superoxide dismutase in vertebrate cells.Pigment epithelium-derived factor delays cellular senescence of human mesenchymal stem cells in vitro by reducing oxidative stress.
P2860
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P2860
Relief of oxidative stress by ascorbic acid delays cellular senescence of normal human and Werner syndrome fibroblast cells.
description
2003 nî lūn-bûn
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2003年の論文
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2003年学术文章
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2003年学术文章
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2003年学术文章
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2003年学术文章
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name
Relief of oxidative stress by ...... ner syndrome fibroblast cells.
@en
Relief of oxidative stress by ...... ner syndrome fibroblast cells.
@nl
type
label
Relief of oxidative stress by ...... ner syndrome fibroblast cells.
@en
Relief of oxidative stress by ...... ner syndrome fibroblast cells.
@nl
prefLabel
Relief of oxidative stress by ...... ner syndrome fibroblast cells.
@en
Relief of oxidative stress by ...... ner syndrome fibroblast cells.
@nl
P2093
P1476
Relief of oxidative stress by ...... ner syndrome fibroblast cells.
@en
P2093
Genro Kashino
Kazuaki Fukase
Keiji Suzuki
Makoto Goto
Masami Watanabe
Seiji Kodama
Yukiko Nakayama
P304
P356
10.1016/S0891-5849(03)00326-5
P577
2003-08-01T00:00:00Z