Pathophysiology of joint pain. - Wikidata - wikimedia - TriplyDB

Pathophysiology of joint pain.

Pathophysiology of joint pain.

http://www.wikidata.org/entity/Q39423228

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Pathophysiology of bone pain: a review.Epidural dexamethasone decreased inflammatory hyperalgesia and spinal cPLA₂ expression in a rat formalin test Reduced cortical responses to noxious heat in patients with rheumatoid arthritis.Putative analgesic activity of repeated oral doses of vitamin E in the treatment of rheumatoid arthritis. Results of a prospective placebo controlled double blind trial NEUROPEPTIDES AND NEUROGENIC MECHANISMS IN ORAL AND PERIODONTAL INFLAMMATION.Breakthrough pain in malignant and non-malignant diseases: a review of prevalence, characteristics and mechanisms.The prostaglandin E2 receptor, EP2, is upregulated in the dorsal root ganglion after painful cervical facet joint injury in the rat.Ketorolac reduces spinal astrocytic activation and PAR1 expression associated with attenuation of pain after facet joint injury.Ablating spinal NK1-bearing neurons eliminates the development of pain and reduces spinal neuronal hyperexcitability and inflammation from mechanical joint injury in the rat.Intra-articular facet joint injections for low back pain: a systematic review.Intensity coding by TMJ-responsive neurons in superficial laminae of caudal medullary dorsal horn of the rat.Response properties of TMJ units in superficial laminae at the spinomedullary junction of female rats vary over the estrous cycle.

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P2860

Pathophysiology of joint pain.

http://www.wikidata.org/entity/Q39423228

description

1996 nî lūn-bûn

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1996年の論文

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1996年学术文章

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1996年学术文章

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1996年学术文章

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1996年学术文章

@zh-my

1996年学术文章

@zh-sg

1996年學術文章

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1996年學術文章

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1996年學術文章

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name

Pathophysiology of joint pain.

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Pathophysiology of joint pain.

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type

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Pathophysiology of joint pain.

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Pathophysiology of joint pain.

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Pathophysiology of joint pain.

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Pathophysiology of joint pain.

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Annals of the Rheumatic Diseases

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Pathophysiology of joint pain.

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Kidd BL

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Morris VH

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Urban L

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P2860

Central changes in processing of mechanoreceptive input in capsaicin-induced secondary hyperalgesia in humans

Neurogenic hyperalgesia: the search for the primary cutaneous afferent fibers that contribute to capsaicin-induced pain and hyperalgesia

Glutamate and substance P coexist in primary afferent terminals in the superficial laminae of spinal cord.

Opioids from immunocytes interact with receptors on sensory nerves to inhibit nociception in inflammation.

The measurement of capsaicin-sensitive sensory nerve fiber function in elderly patients with pain.

Experimental evidence on the nature of cutaneous hyperalgesia.

Effects of interactions of naturally-occurring neuropeptides on blood flow in the rat knee joint.

Assessment of cutaneous sensory and autonomic axon reflexes in rheumatoid arthritis.

Chemical activation of nociceptive peripheral neurones.

Generation of acute pain: central mechanisms.

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276-283

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10.1136/ARD.55.5.276

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14540673

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8660099

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pathophysiology

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1010160

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