Pathophysiology and Current Clinical Management of Preeclampsia.

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Associating Symptom Phenotype and Genotype in Preeclampsia.Serum and Urine Thioflavin-T-Enhanced Fluorescence in Severe Preeclampsia.Platelets in preeclamptic pregnancies fail to exhibit the decrease in mitochondrial oxygen consumption rate seen in normal pregnancies.

P2860

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P2860

Pathophysiology and Current Clinical Management of Preeclampsia.

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http://www.wikidata.org/entity/Q39423454

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

@zh-hans

2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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2017年學術文章

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name

Pathophysiology and Current Clinical Management of Preeclampsia.

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Pathophysiology and Current Clinical Management of Preeclampsia.

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type

Item

label

Pathophysiology and Current Clinical Management of Preeclampsia.

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Pathophysiology and Current Clinical Management of Preeclampsia.

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prefLabel

Pathophysiology and Current Clinical Management of Preeclampsia.

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Pathophysiology and Current Clinical Management of Preeclampsia.

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P1476

Pathophysiology and Current Clinical Management of Preeclampsia.

@en

P2093

Babbette LaMarca

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Lorena M Amaral

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Michelle Owens

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P2860

Clinical risk factors for pre-eclampsia determined in early pregnancy: systematic review and meta-analysis of large cohort studies

Elucidating immune mechanisms causing hypertension during pregnancy

Considerations for Defining Cytokine Dose, Duration, and Milieu That Are Appropriate for Modeling Chronic Low-Grade Inflammation in Type 2 Diabetes

Role of endothelin in mediating soluble fms-like tyrosine kinase 1-induced hypertension in pregnant rats

WHO analysis of causes of maternal death: a systematic review

Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia

Innate immune system and preeclampsia.

Patients with preeclampsia develop agonistic autoantibodies against the angiotensin AT1 receptor.

Increasing maternal body mass index is associated with systemic inflammation in the mother and the activation of distinct placental inflammatory pathways.

Placental debris, oxidative stress and pre-eclampsia.

P2888

s11906-017-0757-7

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scholarly article

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10.1007/S11906-017-0757-7

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P478

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Kedra Wallace

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2017-08-01T00:00:00Z

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28689331

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pathophysiology

pre-eclampsia

maternal health

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5916784

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Pathophysiology and Current Clinical Management of Preeclampsia.

about

P2860

P2860

Pathophysiology and Current Clinical Management of Preeclampsia.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P433

P478

P50

P577

P698

P921

P932

P356

P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P433

P478

P50

P577

P698

P921

P932