about
A Wnt-Axin2-GSK3beta cascade regulates Snail1 activity in breast cancer cellsInhibiting stemness and invasive properties of glioblastoma tumorsphere by combined treatment with temozolomide and a newly designed biguanide (HL156A).p53 and microRNA-34 are suppressors of canonical Wnt signaling.Intraosseous nerve sheath tumors in the jaws.Effects of calcium phosphate ceramic bone graft materials on permanent teeth eruption in beagles.Niclosamide is a potential therapeutic for familial adenomatosis polyposis by disrupting Axin-GSK3 interaction.Anti-helminthic niclosamide inhibits Ras-driven oncogenic transformation via activation of GSK-3Hybrid odontogenic tumor of calcifying odontogenic cyst and ameloblastic fibroma.Implication of snail in metabolic stress-induced necrosis.New class of microRNA targets containing simultaneous 5'-UTR and 3'-UTR interaction sitesA p53/miRNA-34 axis regulates Snail1-dependent cancer cell epithelial-mesenchymal transition.Periosteal osteosarcoma of the mandible.Nuclear hormone receptor corepressor promotes esophageal cancer cell invasion by transcriptional repression of interferon-γ-inducible protein 10 in a casein kinase 2-dependent manner.Metabolic stress induces a Wnt-dependent cancer stem cell-like state transition.p53 regulates nuclear GSK-3 levels through miR-34-mediated Axin2 suppression in colorectal cancer cells.Mutual regulation between DNA-PKcs and Snail1 leads to increased genomic instability and aggressive tumor characteristics.Snail reprograms glucose metabolism by repressing phosphofructokinase PFKP allowing cancer cell survival under metabolic stress.MiRNA-34 intrinsically links p53 tumor suppressor and Wnt signaling.Catabolic metabolism during cancer EMT.The transcription factor snail regulates osteogenic differentiation by repressing Runx2 expression.The effect of alphaMSH analogues on rat bones.Snail-induced EMT promotes cancer stem cell-like properties in head and neck cancer cells.A platform technique for growth factor delivery with novel mode of action.Molecular recognition of proteolytic activity in metastatic cancer cells using fluorogenic gold nanoprobes.Snail-induced epithelial-mesenchymal transition promotes cancer stem cell-like phenotype in head and neck cancer cells.Consecutive targetable smart nanoprobe for molecular recognition of cytoplasmic microRNA in metastatic breast cancer.Protein kinase casein kinase 2-mediated upregulation of N-cadherin confers anoikis resistance on esophageal carcinoma cells.Anchored proteinase-targetable optomagnetic nanoprobes for molecular imaging of invasive cancer cells.O-GlcNAc protein modification in cancer cells increases in response to glucose deprivation through glycogen degradation.Nuclear localization signals of the E-cadherin transcriptional repressor Snail.Effects of artemisinin and its derivatives on growth inhibition and apoptosis of oral cancer cells.Characterization of newly established oral cancer cell lines derived from six squamous cell carcinoma and two mucoepidermoid carcinoma cells.The myoepithelial cell differentiation of mucoepidermoid carcinoma in a collagen gel-based coculture model.Induction of apoptosis and caspase-3 activation by chemopreventive [6]-paradol and structurally related compounds in KB cells.Reversible SUMOylation of TBL1-TBLR1 regulates β-catenin-mediated Wnt signaling.Role of CK1 in GSK3beta-mediated phosphorylation and degradation of snail.The Pentose Phosphate Pathway as a Potential Target for Cancer Therapy.Natural products used as a chemical library for protein-protein interaction targeted drug discovery.Frequent oncogenic BRAF V600E mutation in odontogenic keratocyst.Targeting mutant KRAS with CRISPR-Cas9 controls tumor growth.
P50
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P50
description
researcher
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wetenschapper
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հետազոտող
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name
Jong In Yook
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Jong In Yook
@en
Jong In Yook
@es
Jong In Yook
@nl
Jong In Yook
@sl
type
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Jong In Yook
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Jong In Yook
@en
Jong In Yook
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Jong In Yook
@nl
Jong In Yook
@sl
prefLabel
Jong In Yook
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Jong In Yook
@en
Jong In Yook
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Jong In Yook
@nl
Jong In Yook
@sl
P106
P31
P496
0000-0002-7318-6112