Akt and RhoA inhibition promotes anoikis of aggregated B16F10 melanoma cells.
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Evaluation of MicroRNAs Regulating Anoikis Pathways and Its Therapeutic PotentialThe anti-tumor NC1 domain of collagen XIX inhibits the FAK/ PI3K/Akt/mTOR signaling pathway through αvβ3 integrin interaction.Resistance to the mTOR inhibitor temsirolimus alters adhesion and migration behavior of renal cell carcinoma cells through an integrin α5- and integrin β3-dependent mechanism.
P2860
Akt and RhoA inhibition promotes anoikis of aggregated B16F10 melanoma cells.
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2012 nî lūn-bûn
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2012年の論文
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Akt and RhoA inhibition promotes anoikis of aggregated B16F10 melanoma cells.
@en
Akt and RhoA inhibition promotes anoikis of aggregated B16F10 melanoma cells.
@nl
type
label
Akt and RhoA inhibition promotes anoikis of aggregated B16F10 melanoma cells.
@en
Akt and RhoA inhibition promotes anoikis of aggregated B16F10 melanoma cells.
@nl
prefLabel
Akt and RhoA inhibition promotes anoikis of aggregated B16F10 melanoma cells.
@en
Akt and RhoA inhibition promotes anoikis of aggregated B16F10 melanoma cells.
@nl
P2860
P356
P1476
Akt and RhoA inhibition promotes anoikis of aggregated B16F10 melanoma cells
@en
P2093
Marie-Danielle Nagel
Muriel Vayssade
P2860
P304
P356
10.1042/CBI20110069
P577
2012-03-01T00:00:00Z