β-catenin and Smad3 regulate the activity and stability of myocardin-related transcription factor during epithelial-myofibroblast transition. - Wikidata - wikimedia - TriplyDB

β-catenin and Smad3 regulate the activity and stability of myocardin-related transcription factor during epithelial-myofibroblast transition.

β-catenin and Smad3 regulate the activity and stability of myocardin-related transcription factor during epithelial-myofibroblast transition.

http://www.wikidata.org/entity/Q39454012

about

Mechanisms of TGFβ-Induced Epithelial-Mesenchymal Transition Signaling in Fibrosis: TGF-β, WNT, and YAP/TAZ Converge EMT and interstitial lung disease: a mysterious relationship Matrix biology of idiopathic pulmonary fibrosis: a workshop report of the national heart, lung, and blood institute Mechanobiology of myofibroblast adhesion in fibrotic cardiac disease Molecular mechanisms of epithelial-mesenchymal transition Context-dependent switch in chemo/mechanotransduction via multilevel crosstalk among cytoskeleton-regulated MRTF and TAZ and TGFβ-regulated Smad3 Framework to function: mechanosensitive regulators of gene transcription The Purα/Purβ single-strand DNA-binding proteins attenuate smooth-muscle actin gene transactivation in myofibroblasts.Cellular mechanosensing: getting to the nucleus of it all.Cell adhesion and shape regulate TGF-beta1-induced epithelial-myofibroblast transition via MRTF-A signaling.Myocardin-related Transcription Factor Regulates Nox4 Protein Expression: LINKING CYTOSKELETAL ORGANIZATION TO REDOX STATE.The cellular mastermind(?)-mechanotransduction and the nucleus.Hyperosmotic stress regulates the distribution and stability of myocardin-related transcription factor, a key modulator of the cytoskeleton.Hic-5 is required for myofibroblast differentiation by regulating mechanically dependent MRTF-A nuclear accumulation Transcriptional control of cardiac fibroblast plasticity.Nuclear translocation of myocardin-related transcription factor-A during transforming growth factor beta-induced epithelial to mesenchymal transition of lens epithelial cells.Cell-cell contact and matrix adhesion promote αSMA expression during TGFβ1-induced epithelial-myofibroblast transition via Notch and MRTF-A Biomechanical regulation of mesenchymal cell function β-Catenin/CBP-Dependent Signaling Regulates TGF-β-Induced Epithelial to Mesenchymal Transition of Lens Epithelial Cells Acute lung injury and fibrosis in a baboon model of Escherichia coli sepsis.The fate of the primary cilium during myofibroblast transition The actin-MRTF-SRF gene regulatory axis and myofibroblast differentiation.Control of osteogenesis by the canonical Wnt and BMP pathways in vivo: cooperation and antagonism between the canonical Wnt and BMP pathways as cells differentiate from osteochondroprogenitors to osteoblasts and osteocytes.Smads and cell fate: distinct roles in specification, development, and tumorigenesis in the testis.Regulation and Relevance of Myofibroblast Responses in Idiopathic Pulmonary Fibrosis.Dynamic Interplay of Smooth Muscle α-Actin Gene-Regulatory Proteins Reflects the Biological Complexity of Myofibroblast Differentiation.Cytoskeletal signaling in TGFβ-induced epithelial-mesenchymal transition.Potential Role of Glycogen Synthase Kinase-3β in Regulation of Myocardin Activity in Human Vascular Smooth Muscle Cells.Phosphorylation acts positively and negatively to regulate MRTF-A subcellular localisation and activity.MRTFs- master regulators of EMT.Differential topical susceptibility to TGFβ in intact and injured regions of the epithelium: key role in myofibroblast transition How do your contacts (or their absence) shape your fate?Mechanoregulation of the Myofibroblast in Wound Contraction, Scarring, and Fibrosis: Opportunities for New Therapeutic Intervention.Loss of SMAD3 Promotes Vascular Remodeling in Pulmonary Arterial Hypertension via MRTF Disinhibition.Stromal-derived IGF2 promotes colon cancer progression via paracrine and autocrine mechanisms.TGF-β1 regulates the expression and transcriptional activity of TAZ protein via a Smad3-independent, myocardin-related transcription factor-mediated mechanism.In vitro loading models for tendon mechanobiology.Entanglement of GSK-3β, β-catenin and TGF-β1 signaling network to regulate myocardial fibrosis.The Wnt-β-catenin signaling regulated MRTF-A transcription to activate migration-related genes in human breast cancer cells.

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β-catenin and Smad3 regulate the activity and stability of myocardin-related transcription factor during epithelial-myofibroblast transition.

http://www.wikidata.org/entity/Q39454012

description

2011 nî lūn-bûn

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2011年の論文

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

@zh-my

2011年学术文章

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2011年學術文章

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2011年學術文章

@zh

2011年學術文章

@zh-hant

name

β-catenin and Smad3 regulate t ...... lial-myofibroblast transition.

@en

β-catenin and Smad3 regulate t ...... lial-myofibroblast transition.

@nl

type

label

β-catenin and Smad3 regulate t ...... lial-myofibroblast transition.

@en

β-catenin and Smad3 regulate t ...... lial-myofibroblast transition.

@nl

prefLabel

β-catenin and Smad3 regulate t ...... lial-myofibroblast transition.

@en

β-catenin and Smad3 regulate t ...... lial-myofibroblast transition.

@nl

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MBC.E11-04-0335

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Molecular Biology of the Cell

P1476

β-catenin and Smad3 regulate t ...... lial-myofibroblast transition.

@en

P2093

András Kapus

http://www.w3.org/2001/XMLSchema#string

András Masszi

http://www.w3.org/2001/XMLSchema#string

Emmanuel Charbonney

http://www.w3.org/2001/XMLSchema#string

Hiroyasu Nakano

http://www.w3.org/2001/XMLSchema#string

Pam Speight

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P2860

Smad3 prevents beta-catenin degradation and facilitates beta-catenin nuclear translocation in chondrocytes

Megakaryoblastic leukemia factor-1 transduces cytoskeletal signals and induces smooth muscle cell differentiation from undifferentiated embryonic stem cells

Potentiation of serum response factor activity by a family of myocardin-related transcription factors.

GSK-3: tricks of the trade for a multi-tasking kinase

The basics of epithelial-mesenchymal transition

Dual roles of myocardin-related transcription factors in epithelial mesenchymal transition via slug induction and actin remodeling

Polarity of response to transforming growth factor-beta1 in proximal tubular epithelial cells is regulated by beta-catenin

TGFbeta and Wnt pathway cross-talk

Identification of a novel transcriptional activator, BSAC, by a functional cloning to inhibit tumor necrosis factor-induced cell death

Smad3 mediates transforming growth factor-beta-induced alpha-smooth muscle actin expression

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4472-4485

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scholarly article

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10.1091/MBC.E11-04-0335

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23

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22

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2011-09-30T00:00:00Z

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21965288

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3226468

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