Effect of sodium valproate administration on brain neprilysin expression and memory in rats. - Wikidata - wikimedia - TriplyDB

Effect of sodium valproate administration on brain neprilysin expression and memory in rats.

Effect of sodium valproate administration on brain neprilysin expression and memory in rats.

http://www.wikidata.org/entity/Q39471250

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The Alzheimer's amyloid-degrading peptidase, neprilysin: can we control it?A Long-Term Treatment with Arachidonyl-2'-Chloroethylamide Combined with Valproate Increases Neurogenesis in a Mouse Pilocarpine Model of Epilepsy Current advances in the treatment of Alzheimer's disease: focused on considerations targeting Aβ and tau.Prenatal Hypoxia in Different Periods of Embryogenesis Differentially Affects Cell Migration, Neuronal Plasticity, and Rat Behavior in Postnatal Ontogenesis Neprilysin and Aβ Clearance: Impact of the APP Intracellular Domain in NEP Regulation and Implications in Alzheimer's Disease Epigenetic modifications of chronic hypoxia-mediated neurodegeneration in Alzheimer's disease.Are amyloid-degrading enzymes viable therapeutic targets in Alzheimer's disease?From synaptic spines to nuclear signaling: nuclear and synaptic actions of the amyloid precursor protein.The amyloid precursor protein: a biochemical enigma in brain development, function and disease.Amyloid-clearing proteins and their epigenetic regulation as a therapeutic target in Alzheimer's disease."Boomerang Neuropathology" of Late-Onset Alzheimer's Disease is Shrouded in Harmful "BDDS": Breathing, Diet, Drinking, and Sleep During Aging.Impact of Insulin Degrading Enzyme and Neprilysin in Alzheimer's Disease Biology: Characterization of Putative Cognates for Therapeutic Applications.Fingolimod Exerts only Temporary Antiepileptogenic Effects but Longer-Lasting Positive Effects on Behavior in the WAG/Rij Rat Absence Epilepsy Model.New Insights into Epigenetic and Pharmacological Regulation of Amyloid-Degrading Enzymes.Hypoxia in CNS Pathologies: Emerging Role of miRNA-Based Neurotherapeutics and Yoga Based Alternative Therapies.Hypoxia Affects Neprilysin Expression Through Caspase Activation and an APP Intracellular Domain-dependent Mechanism.APP intracellular domain derived from amyloidogenic β- and γ-secretase cleavage regulates neprilysin expression.Effects of ageing and experimental diabetes on insulin-degrading enzyme expression in male rat tissues.Protective effect of valproic acid in streptozotocin-induced sporadic Alzheimer's disease mouse model: possible involvement of the cholinergic system.Gender difference in valproic acid-induced neuroprotective effects on APP/PS1 double transgenic mice modeling Alzheimer's disease.Valproate improves memory deficits in an Alzheimer's disease mouse model: investigation of possible mechanisms of action.The Aβ-clearance protein transthyretin, like neprilysin, is epigenetically regulated by the amyloid precursor protein intracellular domain.

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P2860

Effect of sodium valproate administration on brain neprilysin expression and memory in rats.

http://www.wikidata.org/entity/Q39471250

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2011 nî lūn-bûn

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Effect of sodium valproate adm ...... expression and memory in rats.

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Effect of sodium valproate adm ...... expression and memory in rats.

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Effect of sodium valproate adm ...... expression and memory in rats.

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Journal of Molecular Neuroscience

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Effect of sodium valproate adm ...... expression and memory in rats.

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P2093

Alasdair R Pickles

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Anthony J Turner

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Daria I Bagrova

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David I Lewis

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Igor A Zhuravin

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Nadezhda M Dubrovskaya

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Natalia N Nalivaeva

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Natalia Z Makova

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Nikolai D Belyaev

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Svetlana A Plesneva

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P2860

Mediator is a transducer of amyloid-precursor-protein-dependent nuclear signalling

An epigenetic mouse model for molecular and behavioral neuropathologies related to schizophrenia vulnerability.

Naturally secreted oligomers of amyloid beta protein potently inhibit hippocampal long-term potentiation in vivo

Identification of the major Abeta1-42-degrading catabolic pathway in brain parenchyma: suppression leads to biochemical and pathological deposition

Etiology of sporadic Alzheimer's disease: somatostatin, neprilysin, and amyloid beta peptide

Presenilin-dependent transcriptional control of the Abeta-degrading enzyme neprilysin by intracellular domains of betaAPP and APLP

Alzheimer's disease: the amyloid cascade hypothesis

Decreased clearance of CNS beta-amyloid in Alzheimer's disease

Recovery of learning and memory is associated with chromatin remodelling

Estrogen stimulates degradation of beta-amyloid peptide by up-regulating neprilysin.

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