Herpes simplex virus latency: adaptation to the peripheral nervous system. II.
about
Thymidine kinase-negative herpes simplex virus mutants establish latency in mouse trigeminal ganglia but do not reactivate.Localization of cis-acting sequence requirements in the promoter of the latency-associated transcript of herpes simplex virus type 1 required for cell-type-specific activity.A conserved open reading frame that overlaps the herpes simplex virus thymidine kinase gene is important for viral growth in cell cultureRegulation and cell-type-specific activity of a promoter located upstream of the latency-associated transcript of herpes simplex virus type 1.
P2860
Herpes simplex virus latency: adaptation to the peripheral nervous system. II.
description
1985 nî lūn-bûn
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1985年の論文
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name
Herpes simplex virus latency: adaptation to the peripheral nervous system. II.
@en
Herpes simplex virus latency: adaptation to the peripheral nervous system. II.
@nl
type
label
Herpes simplex virus latency: adaptation to the peripheral nervous system. II.
@en
Herpes simplex virus latency: adaptation to the peripheral nervous system. II.
@nl
prefLabel
Herpes simplex virus latency: adaptation to the peripheral nervous system. II.
@en
Herpes simplex virus latency: adaptation to the peripheral nervous system. II.
@nl
P2860
P1433
P1476
Herpes simplex virus latency: adaptation to the peripheral nervous system. II.
@en
P2093
P2860
P304
P356
10.3109/07357908509039799
P577
1985-01-01T00:00:00Z