Neuronal CRTC-1 governs systemic mitochondrial metabolism and lifespan via a catecholamine signal.
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DLK-1, SEK-3 and PMK-3 Are Required for the Life Extension Induced by Mitochondrial Bioenergetic Disruption in C. elegansExercise in an electrotactic flow chamber ameliorates age-related degeneration in Caenorhabditis elegans.Synthetic Ligands of Cannabinoid Receptors Affect Dauer Formation in the Nematode Caenorhabditis elegansOxidative stress-induced mitochondrial dysfunction in a normal colon epithelial cell lineEndoplasmic Reticulum Stress Caused by Lipoprotein Accumulation Suppresses Immunity against Bacterial Pathogens and Contributes to ImmunosenescenceMacronutrients and caloric intake in health and longevityMitochondrial metabolites extend lifespan.Nuclear hormone receptors as mediators of metabolic adaptability following reproductive perturbations.CRTC1 gene is differentially methylated in the human hippocampus in Alzheimer's disease.The Aging EpigenomeOctopamine connects nutrient cues to lipid metabolism upon nutrient deprivation.A tachykinin-like neuroendocrine signalling axis couples central serotonin action and nutrient sensing with peripheral lipid metabolism.Splicing factor 1 modulates dietary restriction and TORC1 pathway longevity in C. elegans.Deconvoluting AMP-activated protein kinase (AMPK) adenine nucleotide binding and sensingCREB Regulates Distinct Adaptive Transcriptional Programs in Astrocytes and Neurons.Fragile lifespan expansion by dietary mitohormesis in C. elegans.Lifespan-regulating genes in C. elegans.5'-AMP-Activated Protein Kinase Signaling in Caenorhabditis elegans.Investigating Connections between Metabolism, Longevity, and Behavior in Caenorhabditis elegans.AMPK as a Pro-longevity Target.Animal Models to Study AMPK.Traditional and novel tools to probe the mitochondrial metabolism in health and disease.Membrane ion Channels and Receptors in Animal lifespan Modulation.Deregulation of CRTCs in Aging and Age-Related Disease Risk.Fasting metabolism modulates the interleukin-12/interleukin-10 cytokine axis.Suppression of transcriptional drift extends C. elegans lifespan by postponing the onset of mortality.Transcription factors CEP-1/p53 and CEH-23 collaborate with AAK-2/AMPK to modulate longevity in Caenorhabditis elegans.Genome-wide RNAi Screen for Fat Regulatory Genes in C. elegans Identifies a Proteostasis-AMPK Axis Critical for Starvation Survival.The ω-3 fatty acid α-linolenic acid extends Caenorhabditis elegans lifespan via NHR-49/PPARα and oxidation to oxylipins.Metabotropic GABA signalling modulates longevity in C. elegans.Octopamine enhances oxidative stress resistance through the fasting-responsive transcription factor DAF-16/FOXO in C. elegans.Dietary Restriction and AMPK Increase Lifespan via Mitochondrial Network and Peroxisome Remodeling.Regulation of reproduction and longevity by nutrient-sensing pathways.NHR-49/HNF4 integrates regulation of fatty acid metabolism with a protective transcriptional response to oxidative stress and fasting.Brain-gut communications via distinct neuroendocrine signals bidirectionally regulate longevity in C. elegans.
P2860
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P2860
Neuronal CRTC-1 governs systemic mitochondrial metabolism and lifespan via a catecholamine signal.
description
2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
@zh-my
2015年学术文章
@zh-sg
2015年學術文章
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2015年學術文章
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name
Neuronal CRTC-1 governs system ...... an via a catecholamine signal.
@en
Neuronal CRTC-1 governs system ...... an via a catecholamine signal.
@nl
type
label
Neuronal CRTC-1 governs system ...... an via a catecholamine signal.
@en
Neuronal CRTC-1 governs system ...... an via a catecholamine signal.
@nl
prefLabel
Neuronal CRTC-1 governs system ...... an via a catecholamine signal.
@en
Neuronal CRTC-1 governs system ...... an via a catecholamine signal.
@nl
P2093
P2860
P50
P1433
P1476
Neuronal CRTC-1 governs system ...... pan via a catecholamine signal
@en
P2093
Heather J M Weir
Ianessa Morantte
Kristopher Burkewitz
Olga R Ilkayeva
P2860
P304
P356
10.1016/J.CELL.2015.02.004
P407
P577
2015-02-01T00:00:00Z