Salmonella host cell invasion emerged by acquisition of a mosaic of separate genetic elements, including Salmonella pathogenicity island 1 (SPI1), SPI5, and sopE2.
about
Structural basis for the reversible activation of a Rho protein by the bacterial toxin SopE.Phages and the evolution of bacterial pathogens: from genomic rearrangements to lysogenic conversionSalmonella effectors within a single pathogenicity island are differentially expressed and translocated by separate type III secretion systemsA Genome-Wide siRNA Screen Implicates Spire1/2 in SipA-Driven Salmonella Typhimurium Host Cell InvasionDNA-binding activities of the HilC and HilD virulence regulatory proteins of Salmonella enterica serovar TyphimuriumHierarchical effector protein transport by the Salmonella Typhimurium SPI-1 type III secretion system.SopE and SopE2 from Salmonella typhimurium activate different sets of RhoGTPases of the host cell.Caspase-1 activation via Rho GTPases: a common theme in mucosal infections?In macrophages, caspase-1 activation by SopE and the type III secretion system-1 of S. typhimurium can proceed in the absence of flagellin.Comparative genomic analysis and virulence differences in closely related salmonella enterica serotype heidelberg isolates from humans, retail meats, and animals.Pilot study to evaluate microarray hybridization as a tool for Salmonella enterica serovar Typhimurium strain differentiation.Protective effect of probiotics on Salmonella infectivity assessed with combined in vitro gut fermentation-cellular models.Pathogenicity islands in bacterial pathogenesisSalmonella enterica serovar Typhimurium binds to HeLa cells via Fim-mediated reversible adhesion and irreversible type three secretion system 1-mediated docking.Amino acids of the bacterial toxin SopE involved in G nucleotide exchange on Cdc42.The apoptogenic toxin AIP56 is a metalloprotease A-B toxin that cleaves NF-κb P65Molecular pathogenesis of Salmonella enterica serotype typhimurium-induced diarrhea.Characterization of the yehUT two-component regulatory system of Salmonella enterica Serovar Typhi and Typhimurium.Differences in Salmonella enterica serovar Typhimurium strain invasiveness are associated with heterogeneity in SPI-1 gene expressionPotential origins and horizontal transfer of type III secretion systems and effectors.Non-typhoidal Salmonella Typhimurium ST313 isolates that cause bacteremia in humans stimulate less inflammasome activation than ST19 isolates associated with gastroenteritisRole of the Salmonella pathogenicity island 1 effector proteins SipA, SopB, SopE, and SopE2 in Salmonella enterica subspecies 1 serovar Typhimurium colitis in streptomycin-pretreated mice.Bistable expression of CsgD in Salmonella enterica serovar Typhimurium connects virulence to persistence.The SopEPhi phage integrates into the ssrA gene of Salmonella enterica serovar Typhimurium A36 and is closely related to the Fels-2 prophage.The ancestral SgrS RNA discriminates horizontally acquired Salmonella mRNAs through a single G-U wobble pair.A proteomic approach to the development of DIVA ELISA distinguishing pigs infected with Salmonella Typhimurium and pigs vaccinated with a Salmonella Typhimurium-based inactivated vaccine.Capsule-mediated immune evasion: a new hypothesis explaining aspects of typhoid fever pathogenesisInsertion hot spot for horizontally acquired DNA within a bidirectional small-RNA locus in Salmonella enterica.Salmonella enterica serovar gallinarum requires ppGpp for internalization and survival in animal cellsSubspecies IIIa and IIIb Salmonellae are defective for colonization of murine models of salmonellosis compared to Salmonella enterica subsp. I serovar typhimurium.Mapping the Regulatory Network for Salmonella enterica Serovar Typhimurium Invasion.Salmonellae interactions with host processes.Ubiquitination of the bacterial inositol phosphatase, SopB, regulates its biological activity at the plasma membrane.The Type III Secretion System Effector SeoC of Salmonella enterica subsp. salamae and S. enterica subsp. arizonae ADP-Ribosylates Src and Inhibits Opsonophagocytosis.Life in the inflamed intestine, Salmonella style.The streptomycin mouse model for Salmonella diarrhea: functional analysis of the microbiota, the pathogen's virulence factors, and the host's mucosal immune response.Delineation of the Salmonella enterica serovar Typhimurium HilA regulon through genome-wide location and transcript analysisAnalyses of the evolutionary distribution of Salmonella translocated effectors.The Salmonella enterica serotype typhimurium effector proteins SipA, SopA, SopB, SopD, and SopE2 act in concert to induce diarrhea in calves.Protection of epithelial cells from Salmonella enterica serovar Enteritidis invasion by antibodies against the SPI-1 type III secretion system.
P2860
Q24300730-41406772-8474-48A0-A7DC-97EFF94A0093Q24562822-CC56EC36-724B-4D31-94E9-A669F03403AFQ28490016-DF2A6F4A-99E1-45E4-984F-9D5B3A89DE64Q28554073-335E035A-9D40-4E75-BC67-2589ABD1D6F9Q30309622-9E040AE9-898F-4614-A177-B427C90B6CCEQ30481825-29480AD6-006F-4BC7-AEAA-D8F82B08623EQ33335734-0F463147-94FC-4545-8BE1-95325B0EF74AQ33535907-CFC3E383-61CE-4BCF-8AB8-23D791F79E85Q33683367-00539694-27F2-4408-81C9-0C1750FE9D16Q33696325-ECAB7A43-E9A1-4258-8048-A32DEDA2D9FCQ34041481-4F9450A5-2ACC-45D6-A2E7-3852799F7E31Q34101848-4D768524-00B7-4379-A7EF-4B2189399D6DQ34290188-510D499A-F9B4-4B59-8E90-05D0ACFFA2F4Q34484454-E22AE63F-18AF-4A67-B29E-48FACDF0A75BQ34533184-AE5CA9A5-5ADA-461A-A33F-CCE3AFA753FAQ34611109-005B38A1-6973-4014-B185-985A8D9633DAQ35032182-837F7670-1527-4B05-9492-62D66E3A5DDCQ35080979-17BA8864-8CD0-4024-83F7-06F0BA847D20Q35199123-86128477-2AF6-4A58-827E-3DB88F9580CEQ35305175-51850CA2-8F51-4EE0-B412-BF095B5FD630Q35443593-47777E7C-F892-4961-9C0E-A8A705794ED6Q35549925-EAE3CC27-A303-46CB-A45E-D8B32E351062Q35609348-CF8809C5-3D5E-4AB9-8BB3-216011221112Q35663112-A85C3D7D-43F2-4ED5-B45F-3F8A2C4D3885Q35882335-4E06CDE2-92F6-4662-A712-F0205653EF18Q36190309-BD2677FA-72F4-40F3-AF35-27D422A2219CQ36348390-894FFE9D-D4B5-470E-9B1E-78234ECD8C62Q36672919-A3630DA9-F9B6-45F3-84F8-C12F3BE03533Q36933192-6E56E19C-242E-4E83-B472-C040DD3E2635Q37157045-F40DB1FB-77DD-49BF-AD79-5C9279B3D00FQ37236308-2933317C-49D6-45CA-94A3-0BCC097D27A6Q37357805-3C83FC12-ED43-4C78-BE9B-204F4F3CDD53Q37387728-B4E9DE57-C93D-4BCC-8BB0-8DF7086DB33CQ37425037-29589036-E44B-459A-99BF-23058F553C6FQ37612560-895EF9A2-9947-4BD7-8750-2D6C5D8BEBB1Q37968294-5EBA246F-856A-48A9-9346-7D3A6FCC0E38Q38302051-7C12CCC9-C7E8-438C-A1DB-5D2DCBCB02D6Q39654360-EB11C15F-8EC7-450B-AF7A-B3F9E37E7874Q39655497-C74209F2-FE6C-416C-891E-FAABFF0954A0Q39676097-41DF366B-06AC-42F2-BCB6-3116E181BE4F
P2860
Salmonella host cell invasion emerged by acquisition of a mosaic of separate genetic elements, including Salmonella pathogenicity island 1 (SPI1), SPI5, and sopE2.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh
2001年學術文章
@zh-hant
name
Salmonella host cell invasion ...... and 1 (SPI1), SPI5, and sopE2.
@en
Salmonella host cell invasion ...... and 1 (SPI1), SPI5, and sopE2.
@nl
type
label
Salmonella host cell invasion ...... and 1 (SPI1), SPI5, and sopE2.
@en
Salmonella host cell invasion ...... and 1 (SPI1), SPI5, and sopE2.
@nl
prefLabel
Salmonella host cell invasion ...... and 1 (SPI1), SPI5, and sopE2.
@en
Salmonella host cell invasion ...... and 1 (SPI1), SPI5, and sopE2.
@nl
P2093
P2860
P1476
Salmonella host cell invasion ...... and 1 (SPI1), SPI5, and sopE2.
@en
P2093
A Weissmüller
H Rüssmann
P2860
P304
P356
10.1128/JB.183.7.2348-2358.2001
P407
P577
2001-04-01T00:00:00Z