Deletion of wboA enhances activation of the lectin pathway of complement in Brucella abortus and Brucella melitensis.
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Molecular host-pathogen interaction in brucellosis: current understanding and future approaches to vaccine development for mice and humansRegulation of the mitogen-activated protein kinases by Brucella spp. expressing a smooth and rough phenotype: relationship to pathogen invasiveness.Smooth and rough lipopolysaccharide phenotypes of Brucella induce different intracellular trafficking and cytokine/chemokine release in human monocytes.Immunization of mice with gamma-irradiated Brucella neotomae and its recombinant strains induces protection against virulent B. abortus, B. melitensis, and B. suis challengeBrucella abortus rough mutants induce macrophage oncosis that requires bacterial protein synthesis and direct interaction with the macrophage.Characterization of Brucella abortus O-polysaccharide and core lipopolysaccharide mutants and demonstration that a complete core is required for rough vaccines to be efficient against Brucella abortus and Brucella ovis in the mouse model.The BtaF trimeric autotransporter of Brucella suis is involved in attachment to various surfaces, resistance to serum and virulence.Systems biology analysis of Brucella infected Peyer's patch reveals rapid invasion with modest transient perturbations of the host transcriptome.Lipopolysaccharide-Deficient Brucella Variants Arise Spontaneously during Infection.Role of the Omp25/Omp31 family in outer membrane properties and virulence of Brucella ovisSmooth to Rough Dissociation in Brucella: The Missing Link to VirulenceBrucella spp noncanonical LPS: structure, biosynthesis, and interaction with host immune system.Brucellosis vaccines: assessment of Brucella melitensis lipopolysaccharide rough mutants defective in core and O-polysaccharide synthesis and exportN-Formyl-Perosamine Surface Homopolysaccharides Hinder the Recognition of Brucella abortus by Mouse Neutrophils.An evolutionary strategy for a stealthy intracellular Brucella pathogen.Interactions between Brucella melitensis and human phagocytes: bacterial surface O-Polysaccharide inhibits phagocytosis, bacterial killing, and subsequent host cell apoptosis.Different responses of macrophages to smooth and rough Brucella spp.: relationship to virulenceBrucella abortus rough mutants are cytopathic for macrophages in culture.Brucella melitensis MucR, an orthologue of Sinorhizobium meliloti MucR, is involved in resistance to oxidative, detergent, and saline stresses and cell envelope modifications.Interaction of Brucella suis and Brucella abortus rough strains with human dendritic cells.Chronic brucellosis patients retain low frequency of CD4+ T-lymphocytes expressing CD25 and CD28 after Escherichia coli LPS stimulation of PHA-cultured PBMCs.Single-molecule interrogation of a bacterial sugar transporter allows the discovery of an extracellular inhibitor.Characterization of biological activities of Brucella melitensis lipopolysaccharide.
P2860
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P2860
Deletion of wboA enhances activation of the lectin pathway of complement in Brucella abortus and Brucella melitensis.
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2001 nî lūn-bûn
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2001年の論文
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name
Deletion of wboA enhances acti ...... ortus and Brucella melitensis.
@en
Deletion of wboA enhances acti ...... ortus and Brucella melitensis.
@nl
type
label
Deletion of wboA enhances acti ...... ortus and Brucella melitensis.
@en
Deletion of wboA enhances acti ...... ortus and Brucella melitensis.
@nl
prefLabel
Deletion of wboA enhances acti ...... ortus and Brucella melitensis.
@en
Deletion of wboA enhances acti ...... ortus and Brucella melitensis.
@nl
P2093
P2860
P1476
Deletion of wboA enhances acti ...... ortus and Brucella melitensis.
@en
P2093
Fernandez-Prada CM
Hadfield TL
Nikolich M
Schurig GG
Sriranganathan N
Vemulapalli R
P2860
P304
P356
10.1128/IAI.69.7.4407-4416.2001
P407
P577
2001-07-01T00:00:00Z