Tumor suppressor TNFAIP3 (A20) is frequently deleted in Sézary syndrome.
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Peripheral T-cell and NK-cell lymphomas in the WHO classification: pearls and pitfallsThe role of aberrant proteolysis in lymphomagenesisGenetic rearrangements result in altered gene expression and novel fusion transcripts in Sézary syndromeCharacteristics of CARMA1-BCL10-MALT1-A20-NF-κB expression in T cell-acute lymphocytic leukemiaA20 (TNFAIP3) deletion in Epstein-Barr virus-associated lymphoproliferative disorders/lymphomas.Resistance of Sézary cells to TNF-α-induced apoptosis is mediated in part by a loss of TNFR1 and a high level of the IER3 expressionOverexpression of MALT1-A20-NF-κB in adult B-cell acute lymphoblastic leukemiaComputational Reconstruction of NFκB Pathway Interaction Mechanisms during Prostate Cancer.A20 suppresses hepatocellular carcinoma proliferation and metastasis through inhibition of Twist1 expressionSTX11 functions as a novel tumor suppressor gene in peripheral T-cell lymphomasThe role of A20 in the pathogenesis of lymphocytic malignancy.A20: linking a complex regulator of ubiquitylation to immunity and human diseaseGenomic profiling of Sézary syndrome identifies alterations of key T cell signaling and differentiation genesChanges in the MALT1-A20-NF-κB expression pattern may be related to T cell dysfunction in AML.The TCR γδ repertoire and relative gene expression characteristics of T-ALL cases with biclonal malignant Vδ1 and Vδ2 T cells.Regulation of NF-κB signaling by the A20 deubiquitinase.Regulation of NF-κB by deubiquitinases.Role of the ubiquitin proteasome system in hematologic malignancies.Tumor-suppressor Genes, Cell Cycle Regulatory Checkpoints, and the SkinThe role of the ubiquitin-editing enzyme A20 in diseases of the central nervous system and other pathological processes.MiR-125b regulates proliferation and apoptosis of nasopharyngeal carcinoma by targeting A20/NF-κB signaling pathway.The tumour suppressor p53 is frequently nonfunctional in Sézary syndrome.Genomic landscape of cutaneous T cell lymphoma.Characteristics of A20 gene polymorphisms in T-cell acute lymphocytic leukemia.Different genetic alteration of A20 in a Sézary syndrome case with Vα2-Jα22 T cell clone.Clinicopathologic implication of A20/TNFAIP3 deletion in diffuse large B-cell lymphoma: an analysis according to immunohistochemical subgroups and rituximab treatment.
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P2860
Tumor suppressor TNFAIP3 (A20) is frequently deleted in Sézary syndrome.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Tumor suppressor TNFAIP3 (A20) is frequently deleted in Sézary syndrome.
@en
Tumor suppressor TNFAIP3
@nl
type
label
Tumor suppressor TNFAIP3 (A20) is frequently deleted in Sézary syndrome.
@en
Tumor suppressor TNFAIP3
@nl
prefLabel
Tumor suppressor TNFAIP3 (A20) is frequently deleted in Sézary syndrome.
@en
Tumor suppressor TNFAIP3
@nl
P2093
P2860
P356
P1433
P1476
Tumor suppressor TNFAIP3 (A20) is frequently deleted in Sézary syndrome
@en
P2093
C A Schmidt
F C M Braun
G K Przybylski
J Schröder
P Grabarczyk
P2860
P2888
P304
P356
10.1038/LEU.2011.101
P50
P577
2011-05-31T00:00:00Z