Tumor suppressor TNFAIP3 (A20) is frequently deleted in Sézary syndrome. - Wikidata - wikimedia - TriplyDB

Tumor suppressor TNFAIP3 (A20) is frequently deleted in Sézary syndrome.

Tumor suppressor TNFAIP3 (A20) is frequently deleted in Sézary syndrome.

http://www.wikidata.org/entity/Q39533634

about

Peripheral T-cell and NK-cell lymphomas in the WHO classification: pearls and pitfalls The role of aberrant proteolysis in lymphomagenesis Genetic rearrangements result in altered gene expression and novel fusion transcripts in Sézary syndrome Characteristics of CARMA1-BCL10-MALT1-A20-NF-κB expression in T cell-acute lymphocytic leukemia A20 (TNFAIP3) deletion in Epstein-Barr virus-associated lymphoproliferative disorders/lymphomas.Resistance of Sézary cells to TNF-α-induced apoptosis is mediated in part by a loss of TNFR1 and a high level of the IER3 expression Overexpression of MALT1-A20-NF-κB in adult B-cell acute lymphoblastic leukemia Computational Reconstruction of NFκB Pathway Interaction Mechanisms during Prostate Cancer.A20 suppresses hepatocellular carcinoma proliferation and metastasis through inhibition of Twist1 expression STX11 functions as a novel tumor suppressor gene in peripheral T-cell lymphomas The role of A20 in the pathogenesis of lymphocytic malignancy.A20: linking a complex regulator of ubiquitylation to immunity and human disease Genomic profiling of Sézary syndrome identifies alterations of key T cell signaling and differentiation genes Changes in the MALT1-A20-NF-κB expression pattern may be related to T cell dysfunction in AML.The TCR γδ repertoire and relative gene expression characteristics of T-ALL cases with biclonal malignant Vδ1 and Vδ2 T cells.Regulation of NF-κB signaling by the A20 deubiquitinase.Regulation of NF-κB by deubiquitinases.Role of the ubiquitin proteasome system in hematologic malignancies.Tumor-suppressor Genes, Cell Cycle Regulatory Checkpoints, and the Skin The role of the ubiquitin-editing enzyme A20 in diseases of the central nervous system and other pathological processes.MiR-125b regulates proliferation and apoptosis of nasopharyngeal carcinoma by targeting A20/NF-κB signaling pathway.The tumour suppressor p53 is frequently nonfunctional in Sézary syndrome.Genomic landscape of cutaneous T cell lymphoma.Characteristics of A20 gene polymorphisms in T-cell acute lymphocytic leukemia.Different genetic alteration of A20 in a Sézary syndrome case with Vα2-Jα22 T cell clone.Clinicopathologic implication of A20/TNFAIP3 deletion in diffuse large B-cell lymphoma: an analysis according to immunohistochemical subgroups and rituximab treatment.

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Tumor suppressor TNFAIP3 (A20) is frequently deleted in Sézary syndrome.

http://www.wikidata.org/entity/Q39533634

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

@wuu

2011年论文

@zh

2011年论文

@zh-cn

name

Tumor suppressor TNFAIP3 (A20) is frequently deleted in Sézary syndrome.

@en

Tumor suppressor TNFAIP3

@nl

type

label

Tumor suppressor TNFAIP3 (A20) is frequently deleted in Sézary syndrome.

@en

Tumor suppressor TNFAIP3

@nl

prefLabel

Tumor suppressor TNFAIP3 (A20) is frequently deleted in Sézary syndrome.

@en

Tumor suppressor TNFAIP3

@nl

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Tumor suppressor TNFAIP3 (A20) is frequently deleted in Sézary syndrome

@en

P2093

C A Schmidt

http://www.w3.org/2001/XMLSchema#string

F Busse

http://www.w3.org/2001/XMLSchema#string

F C M Braun

http://www.w3.org/2001/XMLSchema#string

F K Braun

http://www.w3.org/2001/XMLSchema#string

G K Przybylski

http://www.w3.org/2001/XMLSchema#string

J Eberle

http://www.w3.org/2001/XMLSchema#string

J Schröder

http://www.w3.org/2001/XMLSchema#string

M Beyer

http://www.w3.org/2001/XMLSchema#string

M Delin

http://www.w3.org/2001/XMLSchema#string

P Grabarczyk

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P2860

De-ubiquitination and ubiquitin ligase domains of A20 downregulate NF-kappaB signalling

T cell antigen receptor stimulation induces MALT1 paracaspase-mediated cleavage of the NF-kappaB inhibitor A20

Failure to regulate TNF-induced NF-kappaB and cell death responses in A20-deficient mice

Molecular Basis for the Unique Deubiquitinating Activity of the NF-κB Inhibitor A20

The zinc finger protein A20 interacts with a novel anti-apoptotic protein which is cleaved by specific caspases

Recruitment of the IKK signalosome to the p55 TNF receptor: RIP and A20 bind to NEMO (IKKgamma) upon receptor stimulation

TNFAIP3 (A20) is a tumor suppressor gene in Hodgkin lymphoma and primary mediastinal B cell lymphoma.

WHO-EORTC classification for cutaneous lymphomas.

Frequent inactivation of A20 in B-cell lymphomas.

A20 inhibits NF-kappaB activation by dual ubiquitin-editing functions.

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1494-1501

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10.1038/LEU.2011.101

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9

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25

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