NOX2 drives M1-like microglial/macrophage activation and neurodegeneration following experimental traumatic brain injury.
about
NLRP3 Inflammasome in Neurological Diseases, from Functions to TherapiesAcute neuroinflammation induces AIS structural plasticity in a NOX2-dependent manner.Microglial Activation in Traumatic Brain Injury.Microglial-derived microparticles mediate neuroinflammation after traumatic brain injury.NOX2 deficiency alters macrophage phenotype through an IL-10/STAT3 dependent mechanism: implications for traumatic brain injuryMinocycline plus N-acteylcysteine induces remyelination, synergistically protects oligodendrocytes, and modifies neuroinflammation in a rat model of mild traumatic brain injury.The Polarization States of Microglia in TBI: A New Paradigm for Pharmacological Intervention.Microvascular NADPH oxidase in health and disease.NADPH oxidase isoform expression is temporally regulated and may contribute to microglial/macrophage polarization after spinal cord injury.White matter damage after traumatic brain injury: A role for damage associated molecular patterns.NADPH Oxidase 2 Regulates NLRP3 Inflammasome Activation in the Brain after Traumatic Brain Injury.The NADPH oxidase NOX2 mediates loss of parvalbumin interneurons in traumatic brain injury: human autoptic immunohistochemical evidenceQuantitative microglia analyses reveal diverse morphologic responses in the rat cortex after diffuse brain injuryNADPH oxidases in Parkinson's disease: a systematic review.A novel pan-Nox inhibitor, APX-115, protects kidney injury in streptozotocin-induced diabetic mice: possible role of peroxisomal and mitochondrial biogenesis.A combination antioxidant therapy to inhibit NOX2 and activate Nrf2 decreases secondary brain damage and improves functional recovery after traumatic brain injury.Sexual dimorphism in the inflammatory response to traumatic brain injury.MFG-E8 Selectively Inhibited Aβ-Induced Microglial M1 Polarization via NF-κB and PI3K-Akt Pathways.NADPH oxidases in traumatic brain injury - Promising therapeutic targets?A single primary blast-induced traumatic brain injury in rodent model causes cell-type dependent increase in NADPH oxidase isoforms in vulnerable brain regions.Taurine protects dopaminergic neurons in a mouse Parkinson's disease model through inhibition of microglial M1 polarization.Emerging Roles of Immune Cells in Postoperative Cognitive Dysfunction.Combined Blockade of Interleukin-1α and -1β Signaling Protects Mice from Cognitive Dysfunction after Traumatic Brain Injury.Chemopreventive Effects of Phytochemicals and Medicines on M1/M2 Polarized Macrophage Role in Inflammation-Related Diseases
P2860
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P2860
NOX2 drives M1-like microglial/macrophage activation and neurodegeneration following experimental traumatic brain injury.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
NOX2 drives M1-like microglial ...... mental traumatic brain injury.
@en
NOX2 drives M1-like microglial ...... mental traumatic brain injury.
@nl
type
label
NOX2 drives M1-like microglial ...... mental traumatic brain injury.
@en
NOX2 drives M1-like microglial ...... mental traumatic brain injury.
@nl
prefLabel
NOX2 drives M1-like microglial ...... mental traumatic brain injury.
@en
NOX2 drives M1-like microglial ...... mental traumatic brain injury.
@nl
P2093
P2860
P1476
NOX2 drives M1-like microglial ...... imental traumatic brain injury
@en
P2093
Alan I Faden
Alok Kumar
Bogdan A Stoica
Dulce-Mariely Alvarez-Croda
James P Barrett
P2860
P304
P356
10.1016/J.BBI.2016.07.158
P577
2016-07-28T00:00:00Z