Serum-activated K and Cl currents underlay U87-MG glioblastoma cell migration.
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KCa3.1 modulates neuroblast migration along the rostral migratory stream (RMS) in vivo.The inhibition of KCa3.1 channels activity reduces cell motility in glioblastoma derived cancer stem cells.Altered expression of two-pore domain potassium (K2P) channels in cancerCancer as a channelopathy: ion channels and pumps in tumor development and progressionDevelopment of a human extracellular matrix for applications related with stem cells and tissue engineering.Bradykinin-induced chemotaxis of human gliomas requires the activation of KCa3.1 and ClC-3Ionizing radiation, ion transports, and radioresistance of cancer cellsKCa3.1 channels are involved in the infiltrative behavior of glioblastoma in vivo.A proinvasive role for the Ca(2+) -activated K(+) channel KCa3.1 in malignant glioma.Potassium channels in cell cycle and cell proliferation.Cl- and K+ channels and their role in primary brain tumour biology.Ion channels in cancer: future perspectives and clinical potential.Prognostic value of ion channel genes in Chinese patients with gliomas based on mRNA expression profiling.Reconciling the discrepancies on the involvement of large-conductance Ca(2+)-activated K channels in glioblastoma cell migration.KCa3.1 channels and glioblastoma: in vitro studies.KCa3.1 (IK) modulates pancreatic cancer cell migration, invasion and proliferation: anomalous effects on TRAM-34.Overexpression of Large-Conductance Calcium-Activated Potassium Channels in Human Glioblastoma Stem-Like Cells and Their Role in Cell Migration.Hypoxia Modulates the Swelling-Activated Cl Current in Human Glioblastoma Cells: Role in Volume Regulation and Cell Survival.K+ channel signaling in irradiated tumor cells.Cell surface area and membrane folding in glioblastoma cell lines differing in PTEN and p53 status.Identification of key signaling molecules involved in the activation of the swelling-activated chloride current in human glioblastoma cells.Functional roles of the Ca2+-activated K+ channel, KCa3.1, in brain tumors.The role of ion channels in the hypoxia-induced aggressiveness of glioblastoma.Expression and Role of the Intermediate-Conductance Calcium-Activated Potassium Channel KCa3.1 in Glioblastoma.Structure, gating and basic functions of the Ca2+-activated K channel of intermediate conductance.
P2860
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P2860
Serum-activated K and Cl currents underlay U87-MG glioblastoma cell migration.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Serum-activated K and Cl currents underlay U87-MG glioblastoma cell migration.
@en
Serum-activated K and Cl currents underlay U87-MG glioblastoma cell migration.
@nl
type
label
Serum-activated K and Cl currents underlay U87-MG glioblastoma cell migration.
@en
Serum-activated K and Cl currents underlay U87-MG glioblastoma cell migration.
@nl
prefLabel
Serum-activated K and Cl currents underlay U87-MG glioblastoma cell migration.
@en
Serum-activated K and Cl currents underlay U87-MG glioblastoma cell migration.
@nl
P2093
P2860
P50
P356
P1476
Serum-activated K and Cl currents underlay U87-MG glioblastoma cell migration
@en
P2093
Ada Maria Tata
Emilia Castigli
Fabio Franciolini
Francesco Aiello
Paola Ruggieri
P2860
P304
P356
10.1002/JCP.22523
P577
2011-07-01T00:00:00Z