The pathogenic relevance of αM-integrin in Guillain-Barré syndrome.
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Fingolimod attenuates experimental autoimmune neuritis and contributes to Schwann cell-mediated axonal protectionFibronectin connecting segment-1 peptide inhibits pathogenic leukocyte trafficking and inflammatory demyelination in experimental models of chronic inflammatory demyelinating polyradiculoneuropathy.Novel pathomechanisms in inflammatory neuropathies.The Human Blood-Nerve Barrier Transcriptome.
P2860
The pathogenic relevance of αM-integrin in Guillain-Barré syndrome.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
The pathogenic relevance of αM-integrin in Guillain-Barré syndrome.
@en
The pathogenic relevance of αM-integrin in Guillain-Barré syndrome.
@nl
type
label
The pathogenic relevance of αM-integrin in Guillain-Barré syndrome.
@en
The pathogenic relevance of αM-integrin in Guillain-Barré syndrome.
@nl
prefLabel
The pathogenic relevance of αM-integrin in Guillain-Barré syndrome.
@en
The pathogenic relevance of αM-integrin in Guillain-Barré syndrome.
@nl
P2093
P2860
P1476
The pathogenic relevance of αM-integrin in Guillain-Barré syndrome
@en
P2093
Chaoling Dong
Eric Scott Helton
Steven P Palladino
P2860
P2888
P304
P356
10.1007/S00401-016-1599-0
P577
2016-07-26T00:00:00Z