Matrix stiffness modulates proliferation, chemotherapeutic response, and dormancy in hepatocellular carcinoma cells - Wikidata - wikimedia - TriplyDB

Matrix stiffness modulates proliferation, chemotherapeutic response, and dormancy in hepatocellular carcinoma cells

Matrix stiffness modulates proliferation, chemotherapeutic response, and dormancy in hepatocellular carcinoma cells

http://www.wikidata.org/entity/Q39569578

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Tumour progression and metastasis From tissue mechanics to transcription factors.Mechanisms of disseminated cancer cell dormancy: an awakening field Turning hepatic cancer stem cells inside out--a deeper understanding through multiple perspectives.The role of tumor microenvironment in therapeutic resistance EMT, CTCs and CSCs in tumor relapse and drug-resistance Higher Matrix Stiffness Upregulates Osteopontin Expression in Hepatocellular Carcinoma Cells Mediated by Integrin β1/GSK3β/β-Catenin Signaling Pathway A FAK-Cas-Rac-lamellipodin signaling module transduces extracellular matrix stiffness into mechanosensitive cell cycling.Matrix rigidity regulates cancer cell growth by modulating cellular metabolism and protein synthesis.Neuropilin-1 stimulates tumor growth by increasing fibronectin fibril assembly in the tumor microenvironment Cyclic stretching of soft substrates induces spreading and growth Compression stiffening of brain and its effect on mechanosensing by glioma cells Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression.Macro- and micro-environmental factors in clinical hepatocellular cancer.In vitro models of tumor vessels and matrix: engineering approaches to investigate transport limitations and drug delivery in cancer.Epigenetic reprogramming modulates malignant properties of human liver cancer.Macroscopic stiffness of breast tumors predicts metastasis.Fibrosis-dependent mechanisms of hepatocarcinogenesis Wnt signaling induces gene expression of factors associated with bone destruction in lung and breast cancer.CD138-negative myeloma cells regulate mechanical properties of bone marrow stromal cells through SDF-1/CXCR4/AKT signaling pathway Extracellular matrix collagen alters cell proliferation and cell cycle progression of human uterine leiomyoma smooth muscle cells The hepatitis B virus-associated tumor microenvironment in hepatocellular carcinoma.Matrix conditions and KLF2-dependent induction of heme oxygenase-1 modulate inhibition of HCV replication by fluvastatin.Translational horizons in the tumor microenvironment: harnessing breakthroughs and targeting cures.Regulation of TGF-β1-Induced Pro-Apoptotic Signaling by Growth Factor Receptors and Extracellular Matrix Receptor Integrins in the Liver.Lung extracellular matrix and fibroblast function.Novel insights into the function and dynamics of extracellular matrix in liver fibrosis.Stem cells in liver diseases and cancer: recent advances on the path to new therapies.Hepatic stellate cells: central modulators of hepatic carcinogenesis.SDF-1α stiffens myeloma bone marrow mesenchymal stromal cells through the activation of RhoA-ROCK-Myosin II.Tissue stiffness regulates serine/arginine-rich protein-mediated splicing of the extra domain B-fibronectin isoform in tumors Fibrogenic cell reversion underlies fibrosis regression in liver.The MAPK MEK1/2-ERK1/2 Pathway and Its Implication in Hepatocyte Cell Cycle Control.Engineering approaches for investigating tumor angiogenesis: exploiting the role of the extracellular matrix Microenvironment rigidity modulates responses to the HER2 receptor tyrosine kinase inhibitor lapatinib via YAP and TAZ transcription factors.Three-dimensional-engineered matrix to study cancer stem cells and tumorsphere formation: effect of matrix modulus.Role of the microenvironment in the pathogenesis and treatment of hepatocellular carcinoma.The Predictive Link between Matrix and Metastasis.Obesity-dependent changes in interstitial ECM mechanics promote breast tumorigenesis.The tumor microenvironment is a dominant force in multidrug resistance.

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P2860

Matrix stiffness modulates proliferation, chemotherapeutic response, and dormancy in hepatocellular carcinoma cells

http://www.wikidata.org/entity/Q39569578

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Matrix stiffness modulates pro ...... hepatocellular carcinoma cells

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Matrix stiffness modulates pro ...... epatocellular carcinoma cells.

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Matrix stiffness modulates pro ...... hepatocellular carcinoma cells

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Matrix stiffness modulates pro ...... epatocellular carcinoma cells.

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Matrix stiffness modulates pro ...... hepatocellular carcinoma cells

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Matrix stiffness modulates pro ...... epatocellular carcinoma cells.

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Matrix stiffness modulates pro ...... hepatocellular carcinoma cells

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P2093

Alexander Quaas

http://www.w3.org/2001/XMLSchema#string

Daniel Benten

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John P Iredale

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Jörg Schrader

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Mariëlle van Deemter

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Rebecca G Wells

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Rebecca L Aucott

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Shaun Walsh

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Timothy T Gordon-Walker

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P2860

Matrix crosslinking forces tumor progression by enhancing integrin signaling

Tensional homeostasis and the malignant phenotype

Hepatocellular carcinoma in cirrhosis: incidence and risk factors

Cell locomotion and focal adhesions are regulated by substrate flexibility

Clinical management of hepatocellular carcinoma. Conclusions of the Barcelona-2000 EASL conference. European Association for the Study of the Liver

The JAK2 inhibitor AZD1480 potently blocks Stat3 signaling and oncogenesis in solid tumors.

Extracellular matrix: a gatekeeper in the transition from dormancy to metastatic growth

Substrate compliance versus ligand density in cell on gel responses

Prospective comparison of transient elastography, Fibrotest, APRI, and liver biopsy for the assessment of fibrosis in chronic hepatitis C.

Extracellular matrix proteins protect small cell lung cancer cells against apoptosis: a mechanism for small cell lung cancer growth and drug resistance in vivo.

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10.1002/HEP.24108

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4

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53

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Stuart J Forbes

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2011-04-01T00:00:00Z

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50869032

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21442631

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