Clearance of Shigella flexneri infection occurs through a nitric oxide-independent mechanism.

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Innate immune responses in children and adults with Shigellosis The bacterial DNA repair protein Mfd confers resistance to the host nitrogen immune response.

P2860

Q39516488-7AA8736A-C11A-47B4-BDC1-C3A953643783 Q39584185-D7F7B114-2E4C-41DC-B8F4-48954A3CDBA9

P2860

Clearance of Shigella flexneri infection occurs through a nitric oxide-independent mechanism.

Item

http://www.wikidata.org/entity/Q39571812

description

1998 nî lūn-bûn

@nan

1998年の論文

@ja

1998年論文

@yue

1998年論文

@zh-hant

1998年論文

@zh-hk

1998年論文

@zh-mo

1998年論文

@zh-tw

1998年论文

@wuu

1998年论文

@zh

1998年论文

@zh-cn

name

Clearance of Shigella flexneri ...... c oxide-independent mechanism.

@en

Clearance of Shigella flexneri ...... c oxide-independent mechanism.

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type

Item

label

Clearance of Shigella flexneri ...... c oxide-independent mechanism.

@en

Clearance of Shigella flexneri ...... c oxide-independent mechanism.

@nl

prefLabel

Clearance of Shigella flexneri ...... c oxide-independent mechanism.

@en

Clearance of Shigella flexneri ...... c oxide-independent mechanism.

@nl

P1476

Clearance of Shigella flexneri ...... c oxide-independent mechanism.

@en

P2093

M B Goldberg

http://www.w3.org/2001/XMLSchema#string

S S Way

http://www.w3.org/2001/XMLSchema#string

P2860

IpaB mediates macrophage apoptosis induced by Shigella flexneri

Effects of nitric oxide synthase inhibitors on murine infection with Mycobacterium tuberculosis

Susceptibility of a panel of virulent strains of Mycobacterium tuberculosis to reactive nitrogen intermediates

Nitric oxide and macrophage function

Killing of virulent Mycobacterium tuberculosis by reactive nitrogen intermediates produced by activated murine macrophages

Identification of nitric oxide synthase as a protective locus against tuberculosis

Inducible nitric oxide synthase does not affect resolution of murine chlamydial genital tract infections or eradication of chlamydiae in primary murine cell culture.

Antibody and cytokine responses in a mouse pulmonary model of Shigella flexneri serotype 2a infection.

Human monocyte-derived macrophages infected with virulent Shigella flexneri in vitro undergo a rapid cytolytic event similar to oncosis but not apoptosis

Nitric oxide produced during murine listeriosis is protective

P304

3012-3016

http://www.w3.org/2001/XMLSchema#string

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scholarly article

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English

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http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

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1998-06-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

9596784

http://www.w3.org/2001/XMLSchema#string

P921

Shigella flexneri

P932

108306

http://www.w3.org/2001/XMLSchema#string

Clearance of Shigella flexneri infection occurs through a nitric oxide-independent mechanism.

about

P2860

P2860

Clearance of Shigella flexneri infection occurs through a nitric oxide-independent mechanism.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P407

P433

P478

P577

P698

P921

P932

P1433

P1476

P2093

P2860

P304

P31

P407

P433

P478

P577

P698

P921

P932