Rep*: a viral element that can partially replace the origin of plasmid DNA synthesis of Epstein-Barr virus.
about
Plasticity of DNA replication initiation in Epstein-Barr virus episomesTranscription profiling of Epstein-Barr virus nuclear antigen (EBNA)-1 expressing cells suggests targeting of chromatin remodeling complexesThe replicator of the Epstein-Barr virus latent cycle origin of DNA replication, oriP, is composed of multiple functional elements.The dyad symmetry element of Epstein-Barr virus is a dominant but dispensable replication origin.Detection of Epstein-Barr virus genomes in peripheral blood B cells from solid-organ transplant recipients by fluorescence in situ hybridization.Identification of properties of the Kaposi's sarcoma-associated herpesvirus latent origin of replication that are essential for the efficient establishment and maintenance of intact plasmids.Human origin recognition complex binds to the region of the latent origin of DNA replication of Epstein-Barr virus.Replication and partitioning of papillomavirus genomes.The plasmid replicon of Epstein-Barr virus: mechanistic insights into efficient, licensed, extrachromosomal replication in human cells.Identifying sites bound by Epstein-Barr virus nuclear antigen 1 (EBNA1) in the human genome: defining a position-weighted matrix to predict sites bound by EBNA1 in viral genomes.Replication of Epstein-Barr viral DNA.Epigenetic regulation of EBV and KSHV latencyApplying horizontal gene transfer phenomena to enhance non-viral gene therapyThe linking regions of EBNA1 are essential for its support of replication and transcription.Establishment of an oriP replicon is dependent upon an infrequent, epigenetic event.The cis-acting family of repeats can inhibit as well as stimulate establishment of an oriP repliconActivation of TRAF5 and TRAF6 signal cascades negatively regulates the latent replication origin of Epstein-Barr virus through p38 mitogen-activated protein kinase.Initiation of DNA replication within oriP is dispensable for stable replication of the latent Epstein-Barr virus chromosome after infection of established cell lines.The plasmid replicon of EBV consists of multiple cis-acting elements that facilitate DNA synthesis by the cell and a viral maintenance elementThe coupling of synthesis and partitioning of EBV's plasmid replicon is revealed in live cells.The latent origin of replication of Epstein-Barr virus directs viral genomes to active regions of the nucleusEssential elements of a licensed, mammalian plasmid origin of DNA synthesis.
P2860
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P2860
Rep*: a viral element that can partially replace the origin of plasmid DNA synthesis of Epstein-Barr virus.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
1998年论文
@zh
1998年论文
@zh-cn
name
Rep*: a viral element that can ...... nthesis of Epstein-Barr virus.
@en
Rep*: a viral element that can ...... nthesis of Epstein-Barr virus.
@nl
type
label
Rep*: a viral element that can ...... nthesis of Epstein-Barr virus.
@en
Rep*: a viral element that can ...... nthesis of Epstein-Barr virus.
@nl
prefLabel
Rep*: a viral element that can ...... nthesis of Epstein-Barr virus.
@en
Rep*: a viral element that can ...... nthesis of Epstein-Barr virus.
@nl
P2860
P1433
P1476
Rep*: a viral element that can ...... ynthesis of Epstein-Barr virus
@en
P2093
A L Kirchmaier
P2860
P304
P407
P577
1998-06-01T00:00:00Z