Regulation of vibrio cholerae genes required for acid tolerance by a member of the "ToxR-like" family of transcriptional regulators.
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Coping with low pH: molecular strategies in neutralophilic bacteriaCrystal structure of the sensory domain ofEscherichia coliCadC, a member of the ToxR-like protein familyThe crystal structure of AphB, a virulence gene activator from Vibrio cholerae, reveals residues that influence its response to oxygen and pHSelective and Efficient Elimination of Vibrio cholerae with a Chemical Modulator that Targets Glucose MetabolismThe LysR-type virulence activator AphB regulates the expression of genes in Vibrio cholerae in response to low pH and anaerobiosisBiochemical basis for activation of virulence genes by bile salts in Vibrio parahaemolyticus.AphB influences acid tolerance of Vibrio vulnificus by activating expression of the positive regulator CadCCrystal Structure of the Regulatory Domain of AphB from Vibrio vulnificus, a Virulence Gene Regulator.Structure-function analysis of the DNA-binding domain of a transmembrane transcriptional activator.Regulation of acetyl-CoA synthetase transcription by the CrbS/R two-component system is conserved in genetically diverse environmental pathogens.Efficient responses to host and bacterial signals during Vibrio cholerae colonizationPhenotypic and genomic analyses of the Mycobacterium avium complex reveal differences in gastrointestinal invasion and genomic composition.CadC activates pH-dependent expression of the Vibrio vulnificus cadBA operon at a distance through direct binding to an upstream regionVibrio cholerae and cholera: out of the water and into the host.Prevalence and molecular characterization of Vibrio cholerae from ice and beverages sold in Jakarta, Indonesia, using most probable number and multiplex PCR.Intestinal Colonization Dynamics of Vibrio cholerae.Transcriptional regulation in the Streptococcus pneumoniae rlrA pathogenicity islet by RlrA.Insights into Vibrio parahaemolyticus CHN25 response to artificial gastric fluid stress by transcriptomic analysis.A low gastric pH mouse model to evaluate live attenuated bacterial vaccines.Imaging live cells at the nanometer-scale with single-molecule microscopy: obstacles and achievements in experiment optimization for microbiology.Roles of the sodium-translocating NADH:quinone oxidoreductase (Na+-NQR) on vibrio cholerae metabolism, motility and osmotic stress resistance.The role of gastric acid in preventing foodborne disease and how bacteria overcome acid conditions.Lysine decarboxylase expression by Vibrio vulnificus is induced by SoxR in response to superoxide stress.Quorum sensing enhances the stress response in Vibrio cholerae.Acid acclimation by Helicobacter pylori.The LysR-type regulator LeuO regulates the acid tolerance response in Vibrio cholerae.The membrane-bound transcriptional regulator CadC is activated by proteolytic cleavage in response to acid stress.ToxR interferes with CRP-dependent transcriptional activation of ompT in Vibrio cholerae.Characterization of Vibrio cholerae outer membrane vesicles as a candidate vaccine for cholera.Homeostasis of glutathione is associated with polyamine-mediated β-lactam susceptibility in Acinetobacter baumannii ATCC 19606Acid stress response in enteropathogenic gammaproteobacteria: an aptitude for survival.Physiology of the Vc-NhaP paralogous group of cation-proton antiporters in Vibrio cholerae.The ToxR-mediated organic acid tolerance response of Vibrio cholerae requires OmpU.Characterizing the hexose-6-phosphate transport system of Vibrio cholerae, a utilization system for carbon and phosphate sources.Vibrio cholerae RND family efflux systems are required for antimicrobial resistance, optimal virulence factor production, and colonization of the infant mouse small intestine.CadC has a global translational effect during acid adaptation in Salmonella enterica serovar Typhimurium.Lysine decarboxylase of Vibrio parahaemolyticus: kinetics of transcription and role in acid resistance.Induction of manganese-containing superoxide dismutase is required for acid tolerance in Vibrio vulnificus.Identification of novel factors involved in colonization and acid tolerance of Vibrio cholerae.The histone-like protein HU has a role in gene expression during the acid adaptation response in Helicobacter pylori.
P2860
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P2860
Regulation of vibrio cholerae genes required for acid tolerance by a member of the "ToxR-like" family of transcriptional regulators.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
2000年论文
@zh
2000年论文
@zh-cn
name
Regulation of vibrio cholerae ...... of transcriptional regulators.
@en
Regulation of vibrio cholerae ...... of transcriptional regulators.
@nl
type
label
Regulation of vibrio cholerae ...... of transcriptional regulators.
@en
Regulation of vibrio cholerae ...... of transcriptional regulators.
@nl
prefLabel
Regulation of vibrio cholerae ...... of transcriptional regulators.
@en
Regulation of vibrio cholerae ...... of transcriptional regulators.
@nl
P2860
P1476
Regulation of vibrio cholerae ...... of transcriptional regulators.
@en
P2093
D S Merrell
P2860
P304
P356
10.1128/JB.182.19.5342-5350.2000
P407
P577
2000-10-01T00:00:00Z