Human parvovirus B19 induces cell cycle arrest at G(2) phase with accumulation of mitotic cyclins.
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Productive parvovirus B19 infection of primary human erythroid progenitor cells at hypoxia is regulated by STAT5A and MEK signaling but not HIFαThe small 11 kDa nonstructural protein of human parvovirus B19 plays a key role in inducing apoptosis during B19 virus infection of primary erythroid progenitor cells.Phosphorylated STAT5 directly facilitates parvovirus B19 DNA replication in human erythroid progenitors through interaction with the MCM complexHuman papillomavirus type 16 E1 E4-induced G2 arrest is associated with cytoplasmic retention of active Cdk1/cyclin B1 complexes.Chipmunk parvovirus is distinct from members in the genus Erythrovirus of the family Parvoviridae.Human parvovirus B19 causes cell cycle arrest of human erythroid progenitors via deregulation of the E2F family of transcription factorsRole of erythropoietin receptor signaling in parvovirus B19 replication in human erythroid progenitor cellsThe Epstein-Barr virus immediate-early protein BZLF1 induces both a G(2) and a mitotic blockIdentification of a G(2) arrest domain in the E1 wedge E4 protein of human papillomavirus type 16.Development of chemiluminescent assays for the quantitative detection and imaging of 5-bromo-2'deoxyuridine-labeled DNA in parvovirus B19-infected cells.Codon optimization of human parvovirus B19 capsid genes greatly increases their expression in nonpermissive cellsNegative regulation of mitotic promoting factor by the checkpoint kinase chk1 in simian virus 40 lytic infection.BCL2A1a over-expression in murine hematopoietic stem and progenitor cells decreases apoptosis and results in hematopoietic transformation.Parvovirus infection-induced DNA damage responseParvovirus infection-induced cell death and cell cycle arrest.Human herpesvirus 6 suppresses T cell proliferation through induction of cell cycle arrest in infected cells in the G2/M phase.Internal polyadenylation of the parvovirus B19 precursor mRNA is regulated by alternative splicing.Human parvovirus B19 VP2 empty capsids bind to human villous trophoblast cells in vitro via the globoside receptor.The putative metal coordination motif in the endonuclease domain of human Parvovirus B19 NS1 is critical for NS1 induced S phase arrest and DNA damageHuman parvovirus B19: a mechanistic overview of infection and DNA replication.Parvovirus B19 Replication and Expression in Differentiating Erythroid Progenitor CellsHuman parvovirus B19 DNA replication induces a DNA damage response that is dispensable for cell cycle arrest at phase G2/MParvovirus B19 NS1 protein induces cell cycle arrest at G2-phase by activating the ATR-CDC25C-CDK1 pathway.Ex vivo-generated CD36+ erythroid progenitors are highly permissive to human parvovirus B19 replicationGeldanamycin, a ligand of heat shock protein 90, inhibits the replication of herpes simplex virus type 1 in vitro.Human parvovirus B19 NS1 protein modulates inflammatory signaling by activation of STAT3/PIAS3 in human endothelial cells.Block to the production of full-length B19 virus transcripts by internal polyadenylation is overcome by replication of the viral genomeHuman parvovirus B19 infection causes cell cycle arrest of human erythroid progenitors at late S phase that favors viral DNA replication.Comparison of the transcription profile of simian parvovirus with that of the human erythrovirus B19 reveals a number of unique features.Chlamydia trachomatis infection induces cleavage of the mitotic cyclin B1Viral genes as oncolytic agents for cancer therapy.Human Parvoviruses.Parvovirus-induced depletion of cyclin B1 prevents mitotic entry of infected cells.Bluetongue virus infection induces aberrant mitosis in mammalian cellsCell cycle perturbations induced by human herpesvirus 6 infection and their effect on virus replication.Antitumor activity of type III interferon alone or in combination with type I interferon against human non-small cell lung cancer.Mechanisms of cell death in canine parvovirus-infected cells provide intuitive insights to developing nanotools for medicine.Through its nonstructural protein NS1, parvovirus H-1 induces apoptosis via accumulation of reactive oxygen species.Bocavirus infection induces mitochondrion-mediated apoptosis and cell cycle arrest at G2/M phase.Human parvovirus B19 nonstructural protein (NS1) induces cell cycle arrest at G(1) phase.
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P2860
Human parvovirus B19 induces cell cycle arrest at G(2) phase with accumulation of mitotic cyclins.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
2001年论文
@zh
2001年论文
@zh-cn
name
Human parvovirus B19 induces cell cycle arrest at G
@nl
Human parvovirus B19 induces c ...... cumulation of mitotic cyclins.
@en
type
label
Human parvovirus B19 induces cell cycle arrest at G
@nl
Human parvovirus B19 induces c ...... cumulation of mitotic cyclins.
@en
prefLabel
Human parvovirus B19 induces cell cycle arrest at G
@nl
Human parvovirus B19 induces c ...... cumulation of mitotic cyclins.
@en
P2093
P2860
P1433
P1476
Human parvovirus B19 induces c ...... cumulation of mitotic cyclins.
@en
P2093
K Sugamura
N Yaegashi
P2860
P304
P356
10.1128/JVI.75.16.7555-7563.2001
P407
P577
2001-08-01T00:00:00Z