Human papillomavirus type 16 E6 induces cervical cancer cell migration through the p53/microRNA-23b/urokinase-type plasminogen activator pathway.
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MicroRNAs Expressed during Viral Infection: Biomarker Potential and Therapeutic ConsiderationsMultistep model of cervical cancer: participation of miRNAs and coding genesMicroRNA-23b regulates cellular architecture and impairs motogenic and invasive phenotypes during cancer progressionDeregulated miRNAs in human cervical cancer: functional importance and potential clinical use.Papillomavirus E6 oncoproteins.MiR-23b controls ALDH1A1 expression in cervical cancer stem cells.miR-375 activates p21 and suppresses telomerase activity by coordinately regulating HPV E6/E7, E6AP, CIP2A, and 14-3-3ζMicroRNA-23b regulates cyclin-dependent kinase-activating kinase complex through cyclin H repression to modulate endothelial transcription and growth under flowA Comprehensive Review of Dysregulated miRNAs Involved in Cervical Cancer.miR-205 expression promotes cell proliferation and migration of human cervical cancer cellsLong non-coding RNA HOTAIR is associated with human cervical cancer progressionSpatiotemporal dynamics of microRNA during epithelial collective cell migration.Identification of new p53 target microRNAs by bioinformatics and functional analysisSmall molecule inhibitors of human papillomavirus protein - protein interactions.HPV16E6-dependent c-fos expression contributes to AP-1 complex formation in SiHa cells.Deregulation of microRNAs Let-7a and miR-21 mediate aberrant STAT3 signaling during human papillomavirus-induced cervical carcinogenesis: role of E6 oncoprotein.Regulation of cellular miRNA expression by human papillomaviruses.miR-375 is down-regulated in squamous cervical cancer and inhibits cell migration and invasion via targeting transcription factor SP1.The role of histologic subtype, p16(INK4a) expression, and presence of human papillomavirus DNA in penile squamous cell carcinomaRecent advances in the study of HPV-associated carcinogenesisMicroRNA-23b Promotes Avian Leukosis Virus Subgroup J (ALV-J) Replication by Targeting IRF1.Up-Regulation of miR-21 Is Associated with Cervicitis and Human Papillomavirus Infection in Cervical Tissues.Families of microRNAs Expressed in Clusters Regulate Cell Signaling in Cervical Cancer.E6/E7 oncoproteins of high risk HPV-16 upregulate MT1-MMP, MMP-2 and MMP-9 and promote the migration of cervical cancer cells.MicroRNA-214 suppresses growth and invasiveness of cervical cancer cells by targeting UDP-N-acetyl-α-D-galactosamine:polypeptide N-acetylgalactosaminyltransferase 7.Differential expression of microRNAs in bovine papillomavirus type 1 transformed equine cells.miR-23b as a potential tumor suppressor and its regulation by DNA methylation in cervical cancerHuman papillomavirus 16 (HPV16) enhances tumor growth and cancer stemness of HPV-negative oral/oropharyngeal squamous cell carcinoma cells via miR-181 regulation.Regulation of p53 expression and apoptosis by vault RNA2-1-5p in cervical cancer cells.Upregulation of microRNA-224 is associated with aggressive progression and poor prognosis in human cervical cancer.Nip the HPV encoded evil in the cancer bud: HPV reshapes TRAILs and signaling landscapesCellular transformation by human papillomaviruses: lessons learned by comparing high- and low-risk virusesRegulation of viral oncogenesis by microRNAs.miR-2861 acts as a tumor suppressor via targeting EGFR/AKT2/CCND1 pathway in cervical cancer induced by human papillomavirus virus 16 E6Serum microRNA expression levels can predict lymph node metastasis in patients with early-stage cervical squamous cell carcinomaMicroRNA-92 regulates cervical tumorigenesis and its expression is upregulated by human papillomavirus-16 E6 in cervical cancer cells.Suppression of iASPP-dependent aggressiveness in cervical cancer through reversal of methylation silencing of microRNA-124.Increased exosomal microRNA-21 and microRNA-146a levels in the cervicovaginal lavage specimens of patients with cervical cancer.Use of Mature miRNA Strand Selection in miRNAs Families in Cervical Cancer Development.New insights into p53 functions through its target microRNAs.
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P2860
Human papillomavirus type 16 E6 induces cervical cancer cell migration through the p53/microRNA-23b/urokinase-type plasminogen activator pathway.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Human papillomavirus type 16 E ...... plasminogen activator pathway.
@en
Human papillomavirus type 16 E ...... plasminogen activator pathway.
@nl
type
label
Human papillomavirus type 16 E ...... plasminogen activator pathway.
@en
Human papillomavirus type 16 E ...... plasminogen activator pathway.
@nl
prefLabel
Human papillomavirus type 16 E ...... plasminogen activator pathway.
@en
Human papillomavirus type 16 E ...... plasminogen activator pathway.
@nl
P2093
P2860
P356
P1433
P1476
Human papillomavirus type 16 E ...... plasminogen activator pathway.
@en
P2093
P2860
P2888
P304
P356
10.1038/ONC.2010.613
P407
P577
2011-01-17T00:00:00Z