Hyperbaric oxygen intervention reduces secondary spinal cord injury in rats via regulation of HMGB1/TLR4/NF-κB signaling pathway.
about
Hyperbaric oxygen therapy of spinal cord injury.Macrophage polarization: a key event in the secondary phase of acute spinal cord injury.HMGB1, TGF-β and NF-κB are associated with chronic allograft nephropathy.Vitamin D reduces inflammatory response in asthmatic mice through HMGB1/TLR4/NF‑κB signaling pathway.Neuroprotective mechanisms of rutin for spinal cord injury through anti-oxidation and anti-inflammation and inhibition of p38 mitogen activated protein kinase pathway.Cell cycle and complement inhibitors may be specific for treatment of spinal cord injury in aged and young mice: Transcriptomic analyses.
P2860
Hyperbaric oxygen intervention reduces secondary spinal cord injury in rats via regulation of HMGB1/TLR4/NF-κB signaling pathway.
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
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2015年论文
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2015年论文
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name
Hyperbaric oxygen intervention ...... /TLR4/NF-κB signaling pathway.
@en
Hyperbaric oxygen intervention ...... /TLR4/NF-κB signaling pathway.
@nl
type
label
Hyperbaric oxygen intervention ...... /TLR4/NF-κB signaling pathway.
@en
Hyperbaric oxygen intervention ...... /TLR4/NF-κB signaling pathway.
@nl
prefLabel
Hyperbaric oxygen intervention ...... /TLR4/NF-κB signaling pathway.
@en
Hyperbaric oxygen intervention ...... /TLR4/NF-κB signaling pathway.
@nl
P2093
P2860
P1476
Hyperbaric oxygen intervention ...... /TLR4/NF-κB signaling pathway.
@en
P2093
P2860
P304
P577
2015-02-01T00:00:00Z