Akt maintains cell size and survival by increasing mTOR-dependent nutrient uptake.
about
SREBP activity is regulated by mTORC1 and contributes to Akt-dependent cell growthThe insulin-like growth factor-I-mTOR signaling pathway induces the mitochondrial pyrimidine nucleotide carrier to promote cell growthErk regulation of pyruvate dehydrogenase flux through PDK4 modulates cell proliferationMetabolic activation-related CD147-CD98 complexBrick by brick: metabolism and tumor cell growthAKT/PKB signaling: navigating downstreamControlling cell division in yeast and animals: does size matter?Novel drugs that target the metabolic reprogramming in renal cell cancerMetabolic, autophagic, and mitophagic activities in cancer initiation and progressionMolecular Imaging of Metabolic Reprograming in Mutant IDH CellsMedical treatment for gastro-entero-pancreatic neuroendocrine tumoursMetabolic regulation of T cell differentiation and functionRapalogs in cancer prevention: anti-aging or anticancer?Leucine and mTORC1: a complex relationshipAutophagy: a targetable linchpin of cancer cell metabolismRole of AKT/mTORC1 pathway in pancreatic β-cell proliferationMetformin promotes tau aggregation and exacerbates abnormal behavior in a mouse model of tauopathyThe Complexity of Targeting PI3K-Akt-mTOR Signalling in Human Acute Myeloid Leukaemia: The Importance of Leukemic Cell Heterogeneity, Neighbouring Mesenchymal Stem Cells and Immunocompetent CellsAdipose tissue dysfunction and its effects on tumor metabolismFeeding an army: The metabolism of T cells in activation, anergy, and exhaustionNuclear targeting of 6-phosphofructo-2-kinase (PFKFB3) increases proliferation via cyclin-dependent kinasesThe PIM kinases in hematological cancersPhenethyl isothiocyanate exhibits antileukemic activity in vitro and in vivo by inactivation of Akt and activation of JNK pathwaysThe serine/threonine kinase Pim-2 is a transcriptionally regulated apoptotic inhibitorRhoB controls Akt trafficking and stage-specific survival of endothelial cells during vascular developmentNephrin and CD2AP associate with phosphoinositide 3-OH kinase and stimulate AKT-dependent signalingDNA damaging agents and p53 do not cause senescence in quiescent cells, while consecutive re-activation of mTOR is associated with conversion to senescenceGenetic polymorphisms of mTOR and cancer risk: a systematic review and updated meta-analysisBidirectional transport of amino acids regulates mTOR and autophagyMyc regulates a transcriptional program that stimulates mitochondrial glutaminolysis and leads to glutamine addictionBeyond aerobic glycolysis: transformed cells can engage in glutamine metabolism that exceeds the requirement for protein and nucleotide synthesisRheb-TOR signaling promotes protein synthesis, but not glucose or amino acid import, in DrosophilaCluster analysis of protein array results via similarity of Gene Ontology annotation.Systems biology of lupus: mapping the impact of genomic and environmental factors on gene expression signatures, cellular signaling, metabolic pathways, hormonal and cytokine imbalance, and selecting targets for treatment.Glutamine metabolism is essential for human cytomegalovirus infectionMechanism of the salutary effects of estrogen on kupffer cell phagocytic capacity following trauma-hemorrhage: pivotal role of Akt activation.Causal reasoning identifies mechanisms of sensitivity for a novel AKT kinase inhibitor, GSK690693.T-cell and B-cell signaling biomarkers and treatment targets in lupus.Akt-dependent cell size regulation by the adhesion molecule on glia occurs independently of phosphatidylinositol 3-kinase and Rheb signaling.Akt and c-Myc differentially activate cellular metabolic programs and prime cells to bioenergetic inhibition.
P2860
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P2860
Akt maintains cell size and survival by increasing mTOR-dependent nutrient uptake.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
2002年论文
@zh
2002年论文
@zh-cn
name
Akt maintains cell size and survival by increasing mTOR-dependent nutrient uptake.
@en
Akt maintains cell size and survival by increasing mTOR-dependent nutrient uptake.
@nl
type
label
Akt maintains cell size and survival by increasing mTOR-dependent nutrient uptake.
@en
Akt maintains cell size and survival by increasing mTOR-dependent nutrient uptake.
@nl
prefLabel
Akt maintains cell size and survival by increasing mTOR-dependent nutrient uptake.
@en
Akt maintains cell size and survival by increasing mTOR-dependent nutrient uptake.
@nl
P2860
P356
P1476
Akt maintains cell size and survival by increasing mTOR-dependent nutrient uptake.
@en
P2093
Aimee L Edinger
Craig B Thompson
P2860
P304
P356
10.1091/MBC.01-12-0584
P577
2002-07-01T00:00:00Z