A high proliferation rate is required for cell reprogramming and maintenance of human embryonic stem cell identity.
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Rationale and Methodology of Reprogramming for Generation of Induced Pluripotent Stem Cells and Induced Neural Progenitor CellsThe cell cycle and pluripotencydp53 Restrains ectopic neural stem cell formation in the Drosophila brain in a non-apoptotic mechanism involving Archipelago and cyclin EGenetic background affects induced pluripotent stem cell generationThe cell cycle as a brake for β-cell regeneration from embryonic stem cellsEmerging Non-Canonical Functions and Regulation by p53: p53 and StemnessSON connects the splicing-regulatory network with pluripotency in human embryonic stem cellsMacroH2A histone variants act as a barrier upon reprogramming towards pluripotencyA virus-free poly-promoter vector induces pluripotency in quiescent bovine cells under chemically defined conditions of dual kinase inhibitionEfficient generation of iPS cells from skeletal muscle stem cellsA balance between elongation and trimming regulates telomere stability in stem cellsJNK/SAPK Signaling Is Essential for Efficient Reprogramming of Human Fibroblasts to Induced Pluripotent Stem CellsSomatic coding mutations in human induced pluripotent stem cellsIdentification of a specific reprogramming-associated epigenetic signature in human induced pluripotent stem cellsStaged miRNA re-regulation patterns during reprogramming.Kinome-wide functional analysis highlights the role of cytoskeletal remodeling in somatic cell reprogramming.Transitions between epithelial and mesenchymal states during cell fate conversions.p53 regulates cell cycle and microRNAs to promote differentiation of human embryonic stem cells.Serum starvation induced cell cycle synchronization facilitates human somatic cells reprogrammingHigh-risk human papillomavirus E6 protein promotes reprogramming of Fanconi anemia patient cells through repression of p53 but does not allow for sustained growth of induced pluripotent stem cells.Generation of a drug-inducible reporter system to study cell reprogramming in human cells.The homeobox gene DLX4 promotes generation of human induced pluripotent stem cells.A miR-590/Acvr2a/Rad51b axis regulates DNA damage repair during mESC proliferationThe dyskerin ribonucleoprotein complex as an OCT4/SOX2 coactivator in embryonic stem cells.Geminin is required for the maintenance of pluripotency.Polycomb protein SCML2 regulates the cell cycle by binding and modulating CDK/CYCLIN/p21 complexes.p53, Stem Cells, and Reprogramming: Tumor Suppression beyond Guarding the Genome.Increased culture density is linked to decelerated proliferation, prolonged G1 phase, and enhanced propensity for differentiation of self-renewing human pluripotent stem cells.Prolonged proteasome inhibition cyclically upregulates Oct3/4 and Nanog gene expression, but reduces induced pluripotent stem cell colony formation.Embryonic stem cell specific "master" replication origins at the heart of the loss of pluripotencycAMP and EPAC Signaling Functionally Replace OCT4 During Induced Pluripotent Stem Cell Reprogramming.The metabolome of induced pluripotent stem cells reveals metabolic changes occurring in somatic cell reprogrammingProgress in understanding reprogramming to the induced pluripotent stateThe return of metabolism: biochemistry and physiology of the pentose phosphate pathwayPhysical Interactions and Functional Coordination between the Core Subunits of Set1/Mll Complexes and the Reprogramming Factors.Limiting replication stress during somatic cell reprogramming reduces genomic instability in induced pluripotent stem cells.ETS-related transcription factors ETV4 and ETV5 are involved in proliferation and induction of differentiation-associated genes in embryonic stem (ES) cellsExcessive Cellular Proliferation Negatively Impacts Reprogramming Efficiency of Human FibroblastsCultured Cells from the Human Oocyte Cumulus Niche Are Efficient Feeders to Propagate Pluripotent Stem CellsPreserved DNA Damage Checkpoint Pathway Protects against Complications in Long-Standing Type 1 Diabetes.
P2860
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P2860
A high proliferation rate is required for cell reprogramming and maintenance of human embryonic stem cell identity.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
A high proliferation rate is r ...... embryonic stem cell identity.
@en
A high proliferation rate is r ...... embryonic stem cell identity.
@nl
type
label
A high proliferation rate is r ...... embryonic stem cell identity.
@en
A high proliferation rate is r ...... embryonic stem cell identity.
@nl
prefLabel
A high proliferation rate is r ...... embryonic stem cell identity.
@en
A high proliferation rate is r ...... embryonic stem cell identity.
@nl
P2093
P2860
P1433
P1476
A high proliferation rate is r ...... embryonic stem cell identity.
@en
P2093
Athanasia D Panopoulos
Aída Herrerías
Inder M Verma
Karl-Dimiter Bissig
Margaret Lutz
Sergio Ruiz
W Travis Berggren
P2860
P356
10.1016/J.CUB.2010.11.049
P407
P577
2010-12-17T00:00:00Z