Antiproliferative factor decreases Akt phosphorylation and alters gene expression via CKAP4 in T24 bladder carcinoma cells.
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Evidence for bladder urothelial pathophysiology in functional bladder disordersErythropoietin: new directions for the nervous systemHow many lives does CLIMP-63 have?A synthetic form of frizzled 8-associated antiproliferative factor enhances p53 stability through USP2a and MDM2A mouse model for interstitial cystitis/painful bladder syndrome based on APF inhibition of bladder epithelial repair: a pilot studyIntegration analysis of quantitative proteomics and transcriptomics data identifies potential targets of frizzled-8 protein-related antiproliferative factor in vivo.EPO relies upon novel signaling of Wnt1 that requires Akt1, FoxO3a, GSK-3β, and β-catenin to foster vascular integrity during experimental diabetes.Translating cell survival and cell longevity into treatment strategies with SIRT1Antiproliferative factor signaling and interstitial cystitis/painful bladder syndromeAntiproliferative factor regulates connective tissue growth factor (CTGF/CCN2) expression in T24 bladder carcinoma cellsWISP1 (CCN4) autoregulates its expression and nuclear trafficking of β-catenin during oxidant stress with limited effects upon neuronal autophagy.Metformin can block precancerous progression to invasive tumors of bladder through inhibiting STAT3-mediated signaling pathwaysTransmembrane domain is crucial to the subcellular localization and function of Myc target 1.The Dickkopf1-cytoskeleton-associated protein 4 axis creates a novel signalling pathway and may represent a molecular target for cancer therapy.CKAP4 is a Dickkopf1 receptor and is involved in tumor progression.CKAP4 inhibited growth and metastasis of hepatocellular carcinoma through regulating EGFR signaling.Potential anticancer activity of myricetin in human T24 bladder cancer cells both in vitro and in vivo.The effect of a novel frizzled 8-related antiproliferative factor on in vitro carcinoma and melanoma cell proliferation and invasion.Abnormal Akt signalling in bladder epithelial cell explants from patients with interstitial cystitis/bladder pain syndrome can be induced by antiproliferative factor treatment of normal bladder cells.Antiproliferative factor (APF) binds specifically to sites within the cytoskeleton-associated protein 4 (CKAP4) extracellular domain.Long-term ketamine abuse induces cystitis in rats by impairing the bladder epithelial barrier.Proteomics profiling of CLL versus healthy B-cells identifies putative therapeutic targets and a subtype-independent signature of spliceosome dysregulation.Prognostic significance of cytoskeleton-associated membrane protein 4 and its palmitoyl acyltransferase DHHC2 in hepatocellular carcinomaCytoskeleton-Associated Protein 4: Functions Beyond the Endoplasmic Reticulum in Physiology and Disease
P2860
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P2860
Antiproliferative factor decreases Akt phosphorylation and alters gene expression via CKAP4 in T24 bladder carcinoma cells.
description
2010 nî lūn-bûn
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2010年の論文
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2010年論文
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2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
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2010年论文
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2010年论文
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2010年论文
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name
Antiproliferative factor decre ...... n T24 bladder carcinoma cells.
@en
Antiproliferative factor decre ...... n T24 bladder carcinoma cells.
@nl
type
label
Antiproliferative factor decre ...... n T24 bladder carcinoma cells.
@en
Antiproliferative factor decre ...... n T24 bladder carcinoma cells.
@nl
prefLabel
Antiproliferative factor decre ...... n T24 bladder carcinoma cells.
@en
Antiproliferative factor decre ...... n T24 bladder carcinoma cells.
@nl
P2093
P2860
P356
P1476
Antiproliferative factor decre ...... n T24 bladder carcinoma cells.
@en
P2093
Chen-Ou Zhang
Hanief M Shahjee
Kristopher R Koch
Susan K Keay
P2860
P2888
P356
10.1186/1756-9966-29-160
P577
2010-12-10T00:00:00Z
P5875
P6179
1052617609