The retinoblastoma tumor suppressor controls androgen signaling and human prostate cancer progression. - Wikidata - wikimedia - TriplyDB

The retinoblastoma tumor suppressor controls androgen signaling and human prostate cancer progression.

The retinoblastoma tumor suppressor controls androgen signaling and human prostate cancer progression.

http://www.wikidata.org/entity/Q39628604

about

Androgen deprivation-induced senescence promotes outgrowth of androgen-refractory prostate cancer cells Retinoblastoma tumor suppressor functions shared by stem cell and cancer cell strategies Transformation from non-small-cell lung cancer to small-cell lung cancer: molecular drivers and cells of origin Newly identified aspects of tumor suppression by RB Emerging players in prostate cancer: long non-coding RNAs Biologic and clinical significance of androgen receptor variants in castration resistant prostate cancer Cellular prostatic acid phosphatase, a PTEN-functional homologue in prostate epithelia, functions as a prostate-specific tumor suppressor A TRIP230-retinoblastoma protein complex regulates hypoxia-inducible factor-1α-mediated transcription and cancer cell invasion Therapeutically activating RB: reestablishing cell cycle control in endocrine therapy-resistant breast cancer.E2F1 promotes angiogenesis through the VEGF-C/VEGFR-3 axis in a feedback loop for cooperative induction of PDGF-B.HES6 drives a critical AR transcriptional programme to induce castration-resistant prostate cancer through activation of an E2F1-mediated cell cycle network.Direct cooperation between androgen receptor and E2F1 reveals a common regulation mechanism for androgen-responsive genes in prostate cells.Identification of a genetic interaction between the tumor suppressor EAF2 and the retinoblastoma protein (Rb) signaling pathway in C. elegans and prostate cancer cells Aggressive variants of castration-resistant prostate cancer The combination of the prodrugs perforin-CEBPD and perforin-granzyme B efficiently enhances the activation of caspase signaling and kills prostate cancer.Adjusting for misclassification in a stratified biomarker clinical trial.MicroRNA-493 suppresses tumor growth, invasion and metastasis of lung cancer by regulating E2F1 Repression of androgen receptor transcription through the E2F1/DNMT1 axis.The meaning of p16(ink4a) expression in tumors: functional significance, clinical associations and future developments Dual role of FoxA1 in androgen receptor binding to chromatin, androgen signalling and prostate cancer Molecular pathways and targets in prostate cancer A Novel Subset of Human Tumors That Simultaneously Overexpress Multiple E2F-responsive Genes Found in Breast, Ovarian, and Prostate Cancers Organoid cultures derived from patients with advanced prostate cancer.Deletion of the olfactomedin 4 gene is associated with progression of human prostate cancer.A three-marker FISH panel detects more genetic aberrations of AR, PTEN and TMPRSS2/ERG in castration-resistant or metastatic prostate cancers than in primary prostate tumors.Carcinoma initiation via RB tumor suppressor inactivation: a versatile approach to epithelial subtype-dependent cancer initiation in diverse tissues.Metastatic progression of prostate cancer and e-cadherin regulation by zeb1 and SRC family kinases.Novel actions of next-generation taxanes benefit advanced stages of prostate cancer.Targeting Rb inactivation in cancers by synthetic lethality.DNA damage induces GDNF secretion in the tumor microenvironment with paracrine effects promoting prostate cancer treatment resistance.Androgen receptor gene expression in prostate cancer is directly suppressed by the androgen receptor through recruitment of lysine-specific demethylase 1.Androgen receptor-driven chromatin looping in prostate cancer.Anti-cancer effects of novel flavonoid vicenin-2 as a single agent and in synergistic combination with docetaxel in prostate cancer Novel retinoblastoma mutation abrogating the interaction to E2F2/3, but not E2F1, led to selective suppression of thyroid tumors.Outsmarting androgen receptor: creative approaches for targeting aberrant androgen signaling in advanced prostate cancer Aggressive prostate cancer is prevented in ERαKO mice and stimulated in ERβKO TRAMP mice Dual targeting of the Akt/mTOR signaling pathway inhibits castration-resistant prostate cancer in a genetically engineered mouse model.Physical and Functional Interactions between ELL2 and RB in the Suppression of Prostate Cancer Cell Proliferation, Migration, and Invasion.Inactivation of the retinoblastoma gene yields a mouse model of malignant colorectal cancer Estrogen receptors β1 and β2 have opposing roles in regulating proliferation and bone metastasis genes in the prostate cancer cell line PC3.

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P2860

The retinoblastoma tumor suppressor controls androgen signaling and human prostate cancer progression.

http://www.wikidata.org/entity/Q39628604

description

2010 nî lūn-bûn

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2010年論文

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name

The retinoblastoma tumor suppr ...... n prostate cancer progression.

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The retinoblastoma tumor suppr ...... n prostate cancer progression.

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The retinoblastoma tumor suppr ...... n prostate cancer progression.

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The retinoblastoma tumor suppr ...... n prostate cancer progression.

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The retinoblastoma tumor suppr ...... n prostate cancer progression.

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The retinoblastoma tumor suppr ...... n prostate cancer progression.

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Journal of Clinical Investigation

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The retinoblastoma tumor suppr ...... n prostate cancer progression.

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P2093

Adam Ertel

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Agnieszka K Witkiewicz

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Ankur Sharma

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Chellappagounder Thangavel

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Clay E S Comstock

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Colm Morrissey

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Erik S Knudsen

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Ilsa Coleman

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Leonard Gomella

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Nigel Clegg

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P2860

Gene expression profiling predicts clinical outcome of prostate cancer

Cell cycle-regulated association of E2F1 and Sp1 is related to their functional interaction

Molecular determinants of resistance to antiandrogen therapy

Cofactor dynamics and sufficiency in estrogen receptor-regulated transcription

Integrative genomic profiling of human prostate cancer

Castration resistance in human prostate cancer is conferred by a frequently occurring androgen receptor splice variant.

A potent transrepression domain in the retinoblastoma protein induces a cell cycle arrest when bound to E2F sites

Transcriptional networks and cellular senescence in human mammary fibroblasts.

Growth suppression by an E2F-binding-defective retinoblastoma protein (RB): contribution from the RB C pocket.

Estrogen receptor status by immunohistochemistry is superior to the ligand-binding assay for predicting response to adjuvant endocrine therapy in breast cancer.

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4478-4492

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10.1172/JCI44239

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12

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120

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Karen E Knudsen

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2010-11-22T00:00:00Z

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21099110

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