RAGE-dependent signaling in microglia contributes to neuroinflammation, Abeta accumulation, and impaired learning/memory in a mouse model of Alzheimer's disease.
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Receptor for advanced glycation end products and its involvement in inflammatory diseasesMicroglia Ontology and SignalingRAGE Expression and ROS Generation in Neurons: Differentiation versus DamageHypertension and Dementia: Epidemiological and Experimental Evidence Revealing a Detrimental RelationshipKynurenine 3-Monooxygenase: An Influential Mediator of NeuropathologyAmyloid β precursor protein as a molecular target for amyloid β--induced neuronal degeneration in Alzheimer's diseaseMicroglia development and functionInsight of brain degenerative protein modifications in the pathology of neurodegeneration and dementia by proteomic profilingImmunohistochemical study of semicarbazide-sensitive amine oxidase/vascular adhesion protein-1 in the hippocampal vasculature: pathological synergy of Alzheimer's disease and diabetes mellitusInflammation-mediated memory dysfunction and effects of a ketogenic diet in a murine model of multiple sclerosisInvolvement of formyl peptide receptors in receptor for advanced glycation end products (RAGE)--and amyloid beta 1-42-induced signal transduction in glial cellsAn aqueous orally active vaccine targeted against a RAGE/AB complex as a novel therapeutic for Alzheimer's diseaseThe multiple faces of RAGE--opportunities for therapeutic intervention in aging and chronic diseaseSterile inflammation: sensing and reacting to damageMicroglial receptor for advanced glycation end product-dependent signal pathway drives beta-amyloid-induced synaptic depression and long-term depression impairment in entorhinal cortex.Receptor for advanced glycation end products (RAGE) on iNKT cells mediates lung ischemia-reperfusion injury.Chromatin decondensation and T cell hyperresponsiveness in diabetes-associated hyperglycemia.LW-AFC, a new formula derived from Liuwei Dihuang decoction, ameliorates behavioral and pathological deterioration via modulating the neuroendocrine-immune system in PrP-hAβPPswe/PS1ΔE9 transgenic mice.Clinical trial of an inhibitor of RAGE-Aβ interactions in Alzheimer disease.Toll-like receptor signaling and stages of addiction.RAGE inhibition in microglia prevents ischemia-dependent synaptic dysfunction in an amyloid-enriched environmentLate-onset dementia: a mosaic of prototypical pathologies modifiable by diet and lifestyle.Receptor for advanced glycation endproduct modulators: a new therapeutic target in Alzheimer's disease.Association of the RAGE G82S polymorphism with Alzheimer's disease.Neurobiology of microglial action in CNS injuries: receptor-mediated signaling mechanisms and functional rolesRAGE expression in human T cells: a link between environmental factors and adaptive immune responses.T cells promote the regeneration of neural precursor cells in the hippocampus of Alzheimer's disease mice.Regulatory Effect of 25-hydroxyvitamin D3 on Nitric Oxide Production in Activated Microglia.The inhalation anesthetic isoflurane increases levels of proinflammatory TNF-α, IL-6, and IL-1β.The evolving biology of microglia in Alzheimer's disease.High-mobility group box-1 impairs memory in mice through both toll-like receptor 4 and Receptor for Advanced Glycation End Products.Diesel exhaust activates and primes microglia: air pollution, neuroinflammation, and regulation of dopaminergic neurotoxicity.Multi-faced neuroprotective effects of Ginsenoside Rg1 in an Alzheimer mouse model.Increased neuronal PreP activity reduces Aβ accumulation, attenuates neuroinflammation and improves mitochondrial and synaptic function in Alzheimer disease's mouse modelGenetic deficiency of neuronal RAGE protects against AGE-induced synaptic injury.The pattern recognition reagents RAGE VC1 and peptide p5 share common binding sites and exhibit specific reactivity with AA amyloid in miceRAGE is a key cellular target for Abeta-induced perturbation in Alzheimer's disease.Glycation exacerbates the neuronal toxicity of β-amyloid.Neuroanatomical correlates of dysglycemia in young children with type 1 diabetes.Increased expression of the receptor for advanced glycation end products in neurons and astrocytes in a triple transgenic mouse model of Alzheimer's disease.
P2860
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P2860
RAGE-dependent signaling in microglia contributes to neuroinflammation, Abeta accumulation, and impaired learning/memory in a mouse model of Alzheimer's disease.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
2009年论文
@zh
2009年论文
@zh-cn
name
RAGE-dependent signaling in mi ...... model of Alzheimer's disease.
@en
RAGE-dependent signaling in mi ...... model of Alzheimer's disease.
@nl
type
label
RAGE-dependent signaling in mi ...... model of Alzheimer's disease.
@en
RAGE-dependent signaling in mi ...... model of Alzheimer's disease.
@nl
prefLabel
RAGE-dependent signaling in mi ...... model of Alzheimer's disease.
@en
RAGE-dependent signaling in mi ...... model of Alzheimer's disease.
@nl
P2093
P2860
P921
P356
P1433
P1476
RAGE-dependent signaling in mi ...... model of Alzheimer's disease.
@en
P2093
Ann Marie Schmidt
David M Stern
Doris Chen
Douglas G Walker
Hongwei Xu
John S Luddy
John X Chen
Lih-Fen Lue
Shifang Yan
P2860
P304
P356
10.1096/FJ.09-139634
P407
P577
2009-11-11T00:00:00Z