β-catenin is constitutively active and increases STAT3 expression/activation in anaplastic lymphoma kinase-positive anaplastic large cell lymphoma.
about
ALK-driven tumors and targeted therapy: focus on crizotinibThe Wnt/β-catenin pathway cross-talks with STAT3 signaling to regulate survival of retinal pigment epithelium cells.Downregulation of miR-200a induces EMT phenotypes and CSC-like signatures through targeting the β-catenin pathway in hepatic oval cells.Inhibition of β-catenin and STAT3 with a curcumin analog suppresses gastric carcinogenesis in vivoUnique genome-wide map of TCF4 and STAT3 targets using ChIP-seq reveals their association with new molecular subtypes of glioblastoma.Critical analysis of the potential for targeting STAT3 in human malignancy.Docosahexaenoic acid inhibits 12-O-tetradecanoylphorbol-13- acetate-induced fascin-1-dependent breast cancer cell migration by suppressing the PKCδ- and Wnt-1/β-catenin-mediated pathwaysWnt/beta-catenin signaling in glioma.The pathobiology of the oncogenic tyrosine kinase NPM-ALK: a brief update.The emerging role of GSK-3β in the pathobiology of classical Hodgkin lymphoma.Molecular genetics of childhood, adolescent and young adult non-Hodgkin lymphoma.The use of cellular thermal shift assay (CETSA) to study Crizotinib resistance in ALK-expressing human cancers.Aberrant expression and biological significance of Sox2, an embryonic stem cell transcriptional factor, in ALK-positive anaplastic large cell lymphoma.A positive feedback loop involving the Wnt/β-catenin/MYC/Sox2 axis defines a highly tumorigenic cell subpopulation in ALK-positive anaplastic large cell lymphomaSTAT3 paradoxically stimulates β-catenin expression but inhibits β-catenin function.High expression of β-catenin contributes to the crizotinib resistant phenotype in the stem-like cell population in neuroblastoma.Novel Molecular Challenges in Targeting Anaplastic Lymphoma Kinase in ALK-Expressing Human Cancers.Wnt/β-Catenin-Promoted Macrophage Alternative Activation Contributes to Kidney Fibrosis.The absence of a novel intron 19-retaining ALK transcript (ALK-I19) and MYCN amplification correlates with an excellent clinical outcome in neuroblastoma patients.
P2860
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P2860
β-catenin is constitutively active and increases STAT3 expression/activation in anaplastic lymphoma kinase-positive anaplastic large cell lymphoma.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
β-catenin is constitutively ac ...... naplastic large cell lymphoma.
@en
β-catenin is constitutively ac ...... naplastic large cell lymphoma.
@nl
type
label
β-catenin is constitutively ac ...... naplastic large cell lymphoma.
@en
β-catenin is constitutively ac ...... naplastic large cell lymphoma.
@nl
prefLabel
β-catenin is constitutively ac ...... naplastic large cell lymphoma.
@en
β-catenin is constitutively ac ...... naplastic large cell lymphoma.
@nl
P2093
P2860
P1433
P1476
β-catenin is constitutively ac ...... naplastic large cell lymphoma.
@en
P2093
Mona Anand
Pascal Gelebart
Raymond Lai
P2860
P304
P356
10.3324/HAEMATOL.2010.027086
P577
2010-10-22T00:00:00Z