Human cytomegalovirus paralyzes macrophage motility through down-regulation of chemokine receptors, reorganization of the cytoskeleton, and release of macrophage migration inhibitory factor.
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Cytomegalovirus-induced immunopathology and its clinical consequencesPreparation of cryofixed cells for improved 3D ultrastructure with scanning transmission electron tomography.Activation of human macrophages by bacterial components relieves the restriction on replication of an interferon-inducing parainfluenza virus 5 (PIV5) P/V mutantThe latency-associated UL138 gene product of human cytomegalovirus sensitizes cells to tumor necrosis factor alpha (TNF-alpha) signaling by upregulating TNF-alpha receptor 1 cell surface expression.Effect of human cytomegalovirus (HCMV) US27 on CXCR4 receptor internalization measured by fluorogen-activating protein (FAP) biosensors.Analysis of protein expression profiles in the thymus of chickens infected with Marek's disease virus.The US27 gene product of human cytomegalovirus enhances signaling of host chemokine receptor CXCR4.Human cytomegalovirus targets different subsets of antigen-presenting cells with pathological consequences for host immunity: implications for immunosuppression, chronic inflammation and autoimmunity.The 'indirect' effects of cytomegalovirus infection.Surrogate markers of infection: interrogation of the immune system.Interplay between human cytomegalovirus and intrinsic/innate host responses: a complex bidirectional relationship.CXCR4: a virus's best friend?Periodontal herpesviruses: prevalence, pathogenicity, systemic risk.Protein pUL128 of human cytomegalovirus is necessary for monocyte infection and blocking of migration.Human cytomegalovirus infection of M1 and M2 macrophages triggers inflammation and autologous T-cell proliferation.Mumps virus inhibits migration of primary human macrophages toward a chemokine gradient through a TNF-alpha dependent mechanism.CMV late phase-induced mTOR activation is essential for efficient virus replication in polarized human macrophages.Analysis of human cytomegalovirus secondary envelopment by advanced electron microscopy.Human Cytomegalovirus UL111A and US27 gene products enhance the CXCL12/CXCR4 signaling axis via distinct mechanisms.New extracellular factors in glioblastoma multiforme development: neurotensin, growth differentiation factor-15, sphingosine-1-phosphate and cytomegalovirus infection.Natural killer cell-mediated response to human cytomegalovirus-infected macrophages is modulated by their functional polarization
P2860
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P2860
Human cytomegalovirus paralyzes macrophage motility through down-regulation of chemokine receptors, reorganization of the cytoskeleton, and release of macrophage migration inhibitory factor.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
2009年论文
@zh
2009年论文
@zh-cn
name
Human cytomegalovirus paralyze ...... e migration inhibitory factor.
@en
Human cytomegalovirus paralyze ...... e migration inhibitory factor.
@nl
type
label
Human cytomegalovirus paralyze ...... e migration inhibitory factor.
@en
Human cytomegalovirus paralyze ...... e migration inhibitory factor.
@nl
prefLabel
Human cytomegalovirus paralyze ...... e migration inhibitory factor.
@en
Human cytomegalovirus paralyze ...... e migration inhibitory factor.
@nl
P2093
P2860
P921
P1476
Human cytomegalovirus paralyze ...... e migration inhibitory factor.
@en
P2093
Giada Frascaroli
Maria Paola Landini
Michael Bacher
Nina Blankenhorn
Richard Bucala
Robert Pretsch
Stefania Varani
Thomas Mertens
P2860
P304
P356
10.4049/JIMMUNOL.182.1.477
P407
P577
2009-01-01T00:00:00Z