Cisplatin sensitivity of testis tumour cells is due to deficiency in interstrand-crosslink repair and low ERCC1-XPF expression.
about
Systems biology of cisplatin resistance: past, present and futureDNA repair endonuclease ERCC1-XPF as a novel therapeutic target to overcome chemoresistance in cancer therapyBPR1K653, a novel Aurora kinase inhibitor, exhibits potent anti-proliferative activity in MDR1 (P-gp170)-mediated multidrug-resistant cancer cellsThe genomic landscape of testicular germ cell tumours: from susceptibility to treatment.Efficacy of targeted drugs in germ cell cancer cell lines with differential cisplatin sensitivity.Drugging the Cancers Addicted to DNA Repair.Heterogeneous nuclear ribonucleoprotein K is associated with poor prognosis and regulates proliferation and apoptosis in bladder cancer.Quantification and expert evaluation of evidence for chemopredictive biomarkers to personalize cancer treatment.Targeting DNA repair pathways for cancer treatment: what's new?Reduced proficiency in homologous recombination underlies the high sensitivity of embryonal carcinoma testicular germ cell tumors to Cisplatin and poly (adp-ribose) polymerase inhibition.Differential contributory roles of nucleotide excision and homologous recombination repair for enhancing cisplatin sensitivity in human ovarian cancer cellsAssociation between ERCC1 and XPA expression and polymorphisms and the response to cisplatin in testicular germ cell tumours.Role of endonucleases XPF and XPG in nucleotide excision repair of platinated DNA and cisplatin/oxaliplatin cytotoxicity.Intensive chemotherapy as salvage treatment for solid tumors: focus on germ cell cancer.ERCC1 function in nuclear excision and interstrand crosslink repair pathways is mediated exclusively by the ERCC1-202 isoformHuman CD4+CD25+ regulatory T cells are sensitive to low dose cyclophosphamide: implications for the immune response.Whole-exome sequencing reveals the mutational spectrum of testicular germ cell tumours.Expression of domains for protein-protein interaction of nucleotide excision repair proteins modifies cancer cell sensitivity to platinum derivatives and genomic stability.A Review of ERCC1 Gene in Bladder Cancer: Implications for Carcinogenesis and Resistance to ChemoradiotherapyExcision Repair Cross-complementation Group 1 is a Prognostic Biomarker in Patients with Colorectal Cancer Receiving Chemotherapy.Carbon ion beam combined with cisplatin effectively disrupts triple negative breast cancer stem-like cells in vitro.Binding interaction of HMGB4 with cisplatin-modified DNA.JWA reverses cisplatin resistance via the CK2-XRCC1 pathway in human gastric cancer cells.Role of mismatch repair proteins in the processing of cisplatin interstrand cross-links.Cancer stem cells and chemoresistance: The smartest survives the raidA quantitative mass spectrometry-based approach for assessing the repair of 8-methoxypsoralen-induced DNA interstrand cross-links and monoadducts in mammalian cells.The timing of caffeic acid treatment with cisplatin determines sensitization or resistance of ovarian carcinoma cell lines.The prognostic value of DNA damage level in peripheral blood lymphocytes of chemotherapy-naïve patients with germ cell cancer.Cisplatin resistance in germ cell tumours: models and mechanisms.Prognostic and predictive factors in patients treated with chemotherapy for advanced urothelial cancer: where do we stand?Higher expression of XPF is a critical factor in intrinsic chemotherapy resistance of human renal cell carcinoma.Curcumin downregulates p38 MAPK-dependent X-ray repair cross-complement group 1 (XRCC1) expression to enhance cisplatin-induced cytotoxicity in human lung cancer cells.Single nucleotide polymorphisms in DNA repair genes and putative cancer risk.An Overview on Predictive Biomarkers of Testicular Germ Cell Tumors.Malignant melanoma cells acquire resistance to DNA interstrand cross-linking chemotherapeutics by p53-triggered upregulation of DDB2/XPC-mediated DNA repair.A role for the base excision repair enzyme NEIL3 in replication-dependent repair of interstrand DNA cross-links derived from psoralen and abasic sites.Cisplatin Toxicity in Dorsal Root Ganglion Neurons Is Relieved by Meclizine via Diminution of Mitochondrial Compromise and Improved Clearance of DNA Damage.Increased expression of DNA repair gene XPF enhances resistance to hydroxycamptothecin in bladder cancer.Pharmacogenomic considerations in the treatment of muscle-invasive bladder cancer.Diagnostic delay in oncology: a case report of metastatic seminoma.
P2860
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P2860
Cisplatin sensitivity of testis tumour cells is due to deficiency in interstrand-crosslink repair and low ERCC1-XPF expression.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
Cisplatin sensitivity of testi ...... and low ERCC1-XPF expression.
@en
Cisplatin sensitivity of testi ...... and low ERCC1-XPF expression.
@nl
type
label
Cisplatin sensitivity of testi ...... and low ERCC1-XPF expression.
@en
Cisplatin sensitivity of testi ...... and low ERCC1-XPF expression.
@nl
prefLabel
Cisplatin sensitivity of testi ...... and low ERCC1-XPF expression.
@en
Cisplatin sensitivity of testi ...... and low ERCC1-XPF expression.
@nl
P2093
P2860
P356
P1433
P1476
Cisplatin sensitivity of testi ...... and low ERCC1-XPF expression.
@en
P2093
Andrea Piée-Staffa
Astrid Schneider
Beate Köberle
Bernd Kaina
Jürgen Thomale
Svetlana Usanova
Ulrike Sied
P2860
P2888
P356
10.1186/1476-4598-9-248
P577
2010-09-16T00:00:00Z
P5875
P6179
1031819131