P53 is necessary for the apoptotic response mediated by a transient increase of Ras activity
about
Persistent nicotine treatment potentiates amplification of the dihydrofolate reductase gene in rat lung epithelial cells as a consequence of Ras activationInduction of apoptosis in cancer: new therapeutic opportunities.Involvement of Rho family GTPases in p19Arf- and p53-mediated proliferation of primary mouse embryonic fibroblasts.Targeting Bcl-2 stability to sensitize cells harboring oncogenic rasProtein kinase Cδ inactivation inhibits cellular proliferation and decreases survival in human neuroendocrine tumors.PI3K Acts in synergy with loss of PKC to elicit apoptosis via the UPR.Modulation of intracellular signaling pathways to induce apoptosis in prostate cancer cells.Long-term exposure to nicotine, via ras pathway, induces cyclin D1 to stimulate G1 cell cycle transition.Ral A, via activating the mitotic checkpoint, sensitizes cells lacking a functional Nf1 to apoptosis in the absence of protein kinase C.
P2860
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P2860
P53 is necessary for the apoptotic response mediated by a transient increase of Ras activity
description
2002 nî lūn-bûn
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2002年の論文
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2002年論文
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2002年論文
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2002年論文
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2002年論文
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2002年論文
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name
P53 is necessary for the apopt ...... sient increase of Ras activity
@en
P53 is necessary for the apopt ...... ient increase of Ras activity.
@nl
type
label
P53 is necessary for the apopt ...... sient increase of Ras activity
@en
P53 is necessary for the apopt ...... ient increase of Ras activity.
@nl
prefLabel
P53 is necessary for the apopt ...... sient increase of Ras activity
@en
P53 is necessary for the apopt ...... ient increase of Ras activity.
@nl
P2093
P2860
P1476
P53 is necessary for the apopt ...... sient increase of Ras activity
@en
P2093
Chang-Yan Chen
Maureen Magut
Peihong Ma
XinBin Chen
P2860
P304
P356
10.1128/MCB.22.9.2928-2938.2002
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P577
2002-05-01T00:00:00Z