Vav Proteins Are Key Regulators of Card9 Signaling for Innate Antifungal Immunity. - Wikidata - wikimedia - TriplyDB

Vav Proteins Are Key Regulators of Card9 Signaling for Innate Antifungal Immunity.

Vav Proteins Are Key Regulators of Card9 Signaling for Innate Antifungal Immunity.

http://www.wikidata.org/entity/Q39674877

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Immunity against fungi.The C-Type Lectin Mincle: Clues for a Role in Crohn's Disease Adjuvant Reaction.Modulating Host Signaling Pathways to Promote Resistance to Infection by Candida albicans.Molecular and physiological roles of the adaptor protein CARD9 in immunity.Regulation of C-Type Lectin Receptor-Mediated Antifungal Immunity.A minimal RNA ligand for potent RIG-I activation in living mice.Flexible Signaling of Myeloid C-Type Lectin Receptors in Immunity and Inflammation.Innate Immunity against Cryptococcus, from Recognition to Elimination.Inherited CARD9 Deficiency: Invasive Disease Caused by Ascomycete Fungi in Previously Healthy Children and Adults High-Throughput Screening Identifies Genes Required for Induction of Macrophage Pyroptosis Human CARD9: A Critical Molecule of Fungal Immune Surveillance

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Vav Proteins Are Key Regulators of Card9 Signaling for Innate Antifungal Immunity.

http://www.wikidata.org/entity/Q39674877

description

2016 nî lūn-bûn

@nan

2016年の論文

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2016年論文

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2016年論文

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2016年論文

@zh-hk

2016年論文

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2016年論文

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2016年论文

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2016年论文

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2016年论文

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name

Vav Proteins Are Key Regulators of Card9 Signaling for Innate Antifungal Immunity.

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Vav Proteins Are Key Regulators of Card9 Signaling for Innate Antifungal Immunity.

@nl

type

label

Vav Proteins Are Key Regulators of Card9 Signaling for Innate Antifungal Immunity.

@en

Vav Proteins Are Key Regulators of Card9 Signaling for Innate Antifungal Immunity.

@nl

prefLabel

Vav Proteins Are Key Regulators of Card9 Signaling for Innate Antifungal Immunity.

@en

Vav Proteins Are Key Regulators of Card9 Signaling for Innate Antifungal Immunity.

@nl

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J.CELREP.2016.11.018

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Vav Proteins Are Key Regulators of Card9 Signaling for Innate Antifungal Immunity.

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Clarissa Prazeres da Costa

http://www.w3.org/2001/XMLSchema#string

Hanna Bergmann

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Konstantin Neumann

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Lesley Vanes

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Mauricio Menacho-Márquez

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Mihai Netea

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Paul-Albert Koenig

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Susanne Roth

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Victor L Tybulewicz

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Vinod Kumar

http://www.w3.org/2001/XMLSchema#string

P2860

CLEC5A is critical for dengue-virus-induced lethal disease

Human dectin-1 deficiency and mucocutaneous fungal infections

A homozygous CARD9 mutation in a family with susceptibility to fungal infections

Vav family exchange factors: an integrated regulatory and functional view

Structural basis for relief of autoinhibition of the Dbl homology domain of proto-oncogene Vav by tyrosine phosphorylation

Pattern recognition receptors and inflammation

Syk kinase-coupled C-type lectin receptors engage protein kinase C-σ to elicit Card9 adaptor-mediated innate immunity

Immune recognition. A new receptor for beta-glucans

Card9 controls a non-TLR signalling pathway for innate anti-fungal immunity

Differential requirement for Malt1 in T and B cell antigen receptor signaling

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2572-2583

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scholarly article

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10.1016/J.CELREP.2016.11.018

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10

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17

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Bianca Habermann

Xosé R. Bustelo

Melissa D. Johnson

Assa Yeroslaviz

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2016-12-01T00:00:00Z

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27926862

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5177621

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