Activation of NF-kappaB is essential for hepatocyte growth factor-mediated proliferation and tubulogenesis. - Wikidata - wikimedia - TriplyDB

Activation of NF-kappaB is essential for hepatocyte growth factor-mediated proliferation and tubulogenesis.

Activation of NF-kappaB is essential for hepatocyte growth factor-mediated proliferation and tubulogenesis.

http://www.wikidata.org/entity/Q39677451

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Understanding and Targeting MET Signaling in Solid Tumors - Are We There Yet?c-Met as a Target for Personalized Therapy MET: a critical player in tumorigenesis and therapeutic target Hepatocyte growth factor inhibits anoikis in head and neck squamous cell carcinoma cells by activation of ERK and Akt signaling independent of NFkappa B Cellular mechanisms underlying the regulation of dendritic development by hepatocyte growth factor PAI-1 plays a protective role in CCl4-induced hepatic fibrosis in mice: role of hepatocyte division.Proliferation and invasion: plasticity in tumor cells.Hepatocyte growth factor preferentially activates the anti-inflammatory arm of NF-κB signaling to induce A20 and protect renal proximal tubular epithelial cells from inflammation.Research resource: Aorta- and liver-specific ERα-binding patterns and gene regulation by estrogen.Delphinidin inhibits cell proliferation and invasion via modulation of Met receptor phosphorylation.Free fatty acid palmitate impairs the vitality and function of cultured human bladder smooth muscle cells.Activation of stem cells in hepatic diseases.Genetic polymorphisms of epidermal growth factor in relation to risk of hepatocellular carcinoma: two case-control studies.Conditional activation of MET in differentiated skeletal muscle induces atrophy.Synthesis of IL-6 by hepatocytes is a normal response to common hepatic stimuli Satellite cell activation on fibers: modeling events in vivo--an invited review.Hepatocyte-specific c-Met deletion disrupts redox homeostasis and sensitizes to Fas-mediated apoptosis.c-Met-mediated endothelial plasticity drives aberrant vascularization and chemoresistance in glioblastoma Gliotoxin-induced changes in rat liver regeneration after partial hepatectomy.Epstein-Barr Virus Infection of Mammary Epithelial Cells Promotes Malignant Transformation.Circulating interleukin-8 and plasminogen activator inhibitor-1 are increased in women with ovarian carcinoma.The Hepatocyte Growth Factor (HGF)/Met Axis: A Neglected Target in the Treatment of Chronic Myeloproliferative Neoplasms?MET and Small-Cell Lung Cancer.NF-κB in cancer therapy.cMET in NSCLC: Can We Cut off the Head of the Hydra? From the Pathway to the Resistance.Reduced expression of adipose triglyceride lipase enhances tumor necrosis factor alpha-induced intercellular adhesion molecule-1 expression in human aortic endothelial cells via protein kinase C-dependent activation of nuclear factor-kappaB.Evidence that TNF-β (lymphotoxin α) can activate the inflammatory environment in human chondrocytes.Gene of the month: HGF.Protein kinase C inhibitor AEB071 targets ocular melanoma harboring GNAQ mutations via effects on the PKC/Erk1/2 and PKC/NF-κB pathways.The BCR-ABL/NF-κB signal transduction network: a long lasting relationship in Philadelphia positive Leukemias Cell lines expressing recombinant transmembrane domain-activated receptor kinases as tools for drug discovery.HAUSP compartmentalization in chronic myeloid leukemia.MET: roles in epithelial-mesenchymal transition and cancer stemness.Characterization and comprehensive proteome profiling of exosomes secreted by hepatocytes Mitogen-inducible gene 6 is an endogenous inhibitor of HGF/Met-induced cell migration and neurite growth.p38 MAPK is involved in activin A- and hepatocyte growth factor-mediated expression of pro-endocrine gene neurogenin 3 in AR42J-B13 cells.Non genomic loss of function of tumor suppressors in CML: BCR-ABL promotes IκBα mediated p53 nuclear exclusion.InlB-mediated Listeria monocytogenes internalization requires a balanced phospholipase D activity maintained through phospho-cofilin.PDGF-BB Preserves Mitochondrial Morphology, Attenuates ROS Production, and Upregulates Neuroglobin in an Astrocytic Model Under Rotenone Insult.Association of growth factors, HIF-1 and NF-κB expression with proteasomes in endometrial cancer.

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P2860

Activation of NF-kappaB is essential for hepatocyte growth factor-mediated proliferation and tubulogenesis.

http://www.wikidata.org/entity/Q39677451

description

2002 nî lūn-bûn

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2002年の論文

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2002年論文

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2002年論文

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2002年论文

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2002年论文

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2002年论文

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name

Activation of NF-kappaB is ess ...... oliferation and tubulogenesis.

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Activation of NF-kappaB is ess ...... oliferation and tubulogenesis.

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type

label

Activation of NF-kappaB is ess ...... oliferation and tubulogenesis.

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Activation of NF-kappaB is ess ...... oliferation and tubulogenesis.

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prefLabel

Activation of NF-kappaB is ess ...... oliferation and tubulogenesis.

@en

Activation of NF-kappaB is ess ...... oliferation and tubulogenesis.

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MCB.22.4.1060-1072.2002

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Molecular and Cellular Biology

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Activation of NF-kappaB is ess ...... oliferation and tubulogenesis.

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Carola Ponzetto

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Markus Müller

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P2860

A phosphatidylinositol 3-kinase/Akt pathway, activated by tumor necrosis factor or interleukin-1, inhibits apoptosis but does not activate NFkappaB in human endothelial cells

The NF-kappa B and I kappa B proteins: new discoveries and insights

Coupling of a signal response domain in I kappa B alpha to multiple pathways for NF-kappa B activation

HGF receptor associates with the anti-apoptotic protein BAG-1 and prevents cell death

Activation of phosphatidylinositol 3-kinase in response to interleukin-1 leads to phosphorylation and activation of the NF-kappaB p65/RelA subunit

The Gab1 PH domain is required for localization of Gab1 at sites of cell-cell contact and epithelial morphogenesis downstream from the met receptor tyrosine kinase

Sustained recruitment of phospholipase C-gamma to Gab1 is required for HGF-induced branching tubulogenesis

Tumor necrosis factor alpha-induced phosphorylation of RelA/p65 on Ser529 is controlled by casein kinase II

NF-kappaB is a target of AKT in anti-apoptotic PDGF signalling

Induction of epithelial tubules by growth factor HGF depends on the STAT pathway

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1060-1072

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10.1128/MCB.22.4.1060-1072.2002

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4

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Alessandro Morotti

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11549194

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