Th2 cell-specific cytokine expression and allergen-induced airway inflammation depend on JunB. - Wikidata - wikimedia - TriplyDB

Th2 cell-specific cytokine expression and allergen-induced airway inflammation depend on JunB.

Th2 cell-specific cytokine expression and allergen-induced airway inflammation depend on JunB.

http://www.wikidata.org/entity/Q39686503

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Lessons learned from gene expression profiling of cutaneous T-cell lymphoma Does antigen masking by ubiquitin chains protect from the development of autoimmune diseases?Ndfip1 protein promotes the function of itch ubiquitin ligase to prevent T cell activation and T helper 2 cell-mediated inflammation An NF-kappaB-dependent role for JunB in the induction of proinflammatory cytokines in LPS-activated bone marrow-derived dendritic cells.JunB is essential for IL-23-dependent pathogenicity of Th17 cells.Foxp3 positive regulatory T cells: a functional regulation by the E3 ubiquitin ligase Itch.Fms-like tyrosine kinase 3 ligand decreases T helper type 17 cells and suppressors of cytokine signaling proteins in the lung of house dust mite-sensitized and -challenged mice.Convergence of Itch-induced ubiquitination with MEKK1-JNK signaling in Th2 tolerance and airway inflammation.Activation of the E3 ubiquitin ligase Itch through a phosphorylation-induced conformational change Increased frequency of dual-positive TH2/TH17 cells in bronchoalveolar lavage fluid characterizes a population of patients with severe asthma.Requirement for the basic helix-loop-helix transcription factor Dec2 in initial TH2 lineage commitment.Genetic disruption of Fra-1 decreases susceptibility to endotoxin-induced acute lung injury and mortality in mice STAT6-dependent regulation of Th9 development.Impairment of organ-specific T cell negative selection by diabetes susceptibility genes: genomic analysis by mRNA profiling CARMA1 controls Th2 cell-specific cytokine expression through regulating JunB and GATA3 transcription factors.ERK1 is important for Th2 differentiation and development of experimental asthma.Signaling pathways in Th2 development.IL-2 and IL-4 stimulate MEK1 expression and contribute to T cell resistance against suppression by TGF-beta and IL-10 in asthma.Batf is important for IL-4 expression in T follicular helper cells.JunB is required for endothelial cell morphogenesis by regulating core-binding factor beta.Immunity by ubiquitylation: a reversible process of modification.Activator protein 1 (Fos/Jun) functions in inflammatory bone and skin disease The E3 ubiquitin ligase Itch in T cell activation, differentiation, and tolerance.Kinetic analysis of genomewide gene expression reveals molecule circuitries that control T cell activation and Th1/2 differentiation.Activating transcription factor 3 is a negative regulator of allergic pulmonary inflammation TEC and MAPK Kinase Signalling Pathways in T helper (TH) cell Development, TH2 Differentiation and Allergic Asthma.Cbl- and Nedd4-family ubiquitin ligases: balancing tolerance and immunity Th2 cells as targets for therapeutic intervention in allergic bronchial asthma.Preventive and therapeutic anti-inflammatory properties of the sesquiterpene alpha-humulene in experimental airways allergic inflammation.Direct, interferon-independent activation of the CXCL10 promoter by NF-κB and interferon regulatory factor 3 during hepatitis C virus infection.Inhibitory effects of human lactoferrin on U14 cervical carcinoma through upregulation of the immune response.E3 ubiquitin ligases in T-cell tolerance.Molecular mechanisms of T-cell tolerance.Anti-inflammatory effects of resveratrol and its potential use in therapy of immune-mediated diseases.T-cell tolerance in cancer.Multifaceted role of the ubiquitin ligase Itch in immune regulation.Treatment of experimental asthma by decoy-mediated local inhibition of activator protein-1.The E3 ligase Itch in immune regulation and beyond.Regulation of IL-4 Expression in Immunity and Diseases.Allergen-Induced IL-6 Regulates IL-9/IL-17A Balance in CD4+ T Cells in Allergic Airway Inflammation.

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P2860

Th2 cell-specific cytokine expression and allergen-induced airway inflammation depend on JunB.

http://www.wikidata.org/entity/Q39686503

description

2002 nî lūn-bûn

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2002年の論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年论文

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2002年论文

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2002年论文

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name

Th2 cell-specific cytokine exp ...... y inflammation depend on JunB.

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Th2 cell-specific cytokine exp ...... y inflammation depend on JunB.

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type

label

Th2 cell-specific cytokine exp ...... y inflammation depend on JunB.

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Th2 cell-specific cytokine exp ...... y inflammation depend on JunB.

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prefLabel

Th2 cell-specific cytokine exp ...... y inflammation depend on JunB.

@en

Th2 cell-specific cytokine exp ...... y inflammation depend on JunB.

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The EMBO Journal

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Th2 cell-specific cytokine exp ...... y inflammation depend on JunB.

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P2093

Bettina Hartenstein

http://www.w3.org/2001/XMLSchema#string

Jochen Hess

http://www.w3.org/2001/XMLSchema#string

Johannes Schenkel

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Marina Schorpp-Kistner

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Peter Angel

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Sibylle Teurich

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P2860

A novel transcription factor, T-bet, directs Th1 lineage commitment

Activation of the interleukin-5 promoter by cAMP in murine EL-4 cells requires the GATA-3 and CLE0 elements

Regulation of IL-4 expression by the transcription factor JunB during T helper cell differentiation

The biology of Stat4 and Stat6

Interleukin-13: central mediator of allergic asthma

The transcription factor c-Maf controls the production of interleukin-4 but not other Th2 cytokines

Inhibition of allergic inflammation in a murine model of asthma by expression of a dominant-negative mutant of GATA-3

JunB can substitute for Jun in mouse development and cell proliferation

JunB is essential for mammalian placentation

The transcription factor GATA-3 is necessary and sufficient for Th2 cytokine gene expression in CD4 T cells

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6321-6329

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scholarly article

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10.1093/EMBOJ/CDF648

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23

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21

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Marina Schorpp-Kistner

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2002-12-01T00:00:00Z

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277498630

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12456639

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136952

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